Giulia Chinetti-Gbaguidi
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Explore the profile of Giulia Chinetti-Gbaguidi including associated specialties, affiliations and a list of published articles.
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41
Citations
2405
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Recent Articles
1.
Lahaye C, Gladine C, Pereira B, Berger J, Chinetti-Gbaguidi G, Laine F, et al.
J Trace Elem Med Biol
. 2021 May;
67:126786.
PMID: 34022567
Aims: Dysmetabolic iron overload syndrome (DIOS) is common but the clinical relevance of iron overload is not understood. Macrophages are central cells in iron homeostasis and inflammation. We hypothesized that...
2.
Chinetti-Gbaguidi G, Daoudi M, Rosa M, Vinod M, Louvet L, Copin C, et al.
Circ Res
. 2017 Apr;
121(1):19-30.
PMID: 28438779
Rationale: Vascular calcification is a process similar to bone formation leading to an inappropriate deposition of calcium phosphate minerals in advanced atherosclerotic plaques. Monocyte-derived macrophages, located in atherosclerotic lesions and...
3.
Ferret-Sena V, Maia E Silva A, Sena A, Cavaleiro I, Vale J, Derudas B, et al.
PPAR Res
. 2017 Jan;
2016:5716415.
PMID: 28077943
Peroxisome Proliferator-Activated Receptors (PPAR) are transcription factors suggested to be involved in inflammatory lesions of autoimmune encephalomyelitis and multiple sclerosis (MS). Our objective was to assess whether Natalizumab (NTZ) therapy...
4.
Bosviel R, Joumard-Cubizolles L, Chinetti-Gbaguidi G, Bayle D, Copin C, Hennuyer N, et al.
Free Radic Biol Med
. 2016 Dec;
103:146-154.
PMID: 27988338
Whereas the anti-inflammatory properties and mechanisms of action of long chain ω3 PUFAs have been abundantly investigated, research gaps remain regarding the respective contribution and mechanisms of action of their...
5.
Bricambert J, Favre D, Brajkovic S, Bonnefond A, Boutry R, Salvi R, et al.
Mol Metab
. 2016 Dec;
5(12):1200-1207.
PMID: 27900262
Objective: The goal of the study was to investigate the role of histone deacetylases (HDACs) in adipocyte function associated with obesity and hypoxia. Methods: Total proteins and RNA were prepared...
6.
De Paoli F, Copin C, Vanhoutte J, Derudas B, Vinod M, Zawadzki C, et al.
FEBS Lett
. 2016 Jan;
590(1):43-52.
PMID: 26763127
Macrophages display heterogeneous phenotypes, including the classical M1 proinflammatory and the alternative M2 anti-inflammatory polarization states. The transducin-like enhancer of split-1 (TLE1) is a transcriptional corepressor whose functions in macrophages...
7.
Roma-Lavisse C, Tagzirt M, Zawadzki C, Lorenzi R, Vincentelli A, Haulon S, et al.
Diab Vasc Dis Res
. 2015 May;
12(4):279-89.
PMID: 25966737
This study aimed to investigate atherosclerotic mediators' expression levels in M1 and M2 macrophages and to focus on the influence of diabetes on M1/M2 profiles. Macrophages from 36 atherosclerotic patients...
8.
Colin S, Chinetti-Gbaguidi G, Kuivenhoven J, Staels B
Handb Exp Pharmacol
. 2014 Dec;
224:617-30.
PMID: 25523004
Dyslipidaemia is a major risk factor for cardiovascular diseases. Pharmacological lowering of LDL-C levels using statins reduces cardiovascular risk. However, a substantial residual risk persists especially in patients with type...
9.
Chinetti-Gbaguidi G, Colin S, Staels B
Nat Rev Cardiol
. 2014 Nov;
12(1):10-7.
PMID: 25367649
Macrophage accumulation within the vascular wall is a hallmark of atherosclerosis. In atherosclerotic lesions, macrophages respond to various environmental stimuli, such as modified lipids, cytokines, and senescent erythrocytes, which can...
10.
Colin S, Chinetti-Gbaguidi G, Staels B
Immunol Rev
. 2014 Oct;
262(1):153-66.
PMID: 25319333
Initiation and progression of atherosclerosis depend on local inflammation and accumulation of lipids in the vascular wall. Although many cells are involved in the development and progression of atherosclerosis, macrophages...