G T Elliott
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Explore the profile of G T Elliott including associated specialties, affiliations and a list of published articles.
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37
Citations
147
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Recent Articles
1.
Okubo S, Bernardo N, Elliott G, Hess M, Kukreja R
Am J Physiol Heart Circ Physiol
. 2000 Oct;
279(5):H2269-76.
PMID: 11045962
We investigated the role of tyrosine kinase (TK) signaling in the opening of the ATP-sensitive K(+) (K(ATP)) channel and 72-kDa heat shock protein (HSP72) expression during late preconditioning. Rabbits were...
2.
Yoshida T, Engelman R, Engelman D, Rousou J, Maulik N, Sato M, et al.
Ann Thorac Surg
. 2000 Oct;
70(3):895-900.
PMID: 11016330
Background: Ischemic preconditioning has been proven to be a powerful tool for myocardial protection in the setting of ischemia and reperfusion. A new drug to provide pharmacologic preconditioning, monophosphoryl lipid...
3.
Elliott G, Sowell C, Walker E, Weber P, Moore J, Gross G
J Mol Cell Cardiol
. 2000 Jun;
32(7):1327-39.
PMID: 10860773
The novel glycolipid RC-552 shares common structural features with the natural products lipid A and the previously described cardioprotectant monophosphoryl lipid A. RC-552 administered to dogs as a bolus intravenous...
4.
Przyklenk K, Hata K, Whittaker P, Elliott G
J Cardiovasc Pharmacol
. 2000 Mar;
35(3):366-75.
PMID: 10710120
Nitric oxide (NO) is a potent inhibitor of platelet aggregation. However, the benefits of NO-based therapies can be confounded by concomitant hypotension. Monophosphoryl lipid A (MLA) is a nontoxic derivative...
5.
Zhao L, Elliott G
Ann N Y Acad Sci
. 1999 Jul;
874:222-35.
PMID: 10415534
In comparison with ischemic preconditioning, MLA-mediated cardioprotection seems to show numerous common features. Like ischemia, MLA induces a first and second window (biphasic profile) of heightened tolerance to ischemia. As...
6.
Przyklenk K, Simkhovich B, Bauer B, Hata K, Zhao L, Elliott G, et al.
Ann N Y Acad Sci
. 1999 Jul;
874:192-210.
PMID: 10415532
Brief episodes of ischemia protect or "precondition" the heart and reduce infarct size caused by a subsequent sustained ischemic insult. Despite a decade of intensive investigation, the cellular mechanism(s) responsible...
7.
Stambaugh K, Elliott G, Jacobson K, Liang B
Circulation
. 1999 Jun;
99(25):3300-7.
PMID: 10385506
Background: The cardioprotective effect of preconditioning can be exerted within 1 to 2 hours after initial ischemia, termed classical or early preconditioning, or can reappear 24 hours later as second...
8.
Maulik N, Tosaki A, Elliott G, Maulik G, Das D
Drugs Exp Clin Res
. 1998 Nov;
24(3):117-24.
PMID: 9825227
Using the concept that exposing a cell to an adverse environment (stress) results in the stimulation of its endogenous defense system, hearts have been adapted to ischemia by exposing them...
9.
Janin Y, Qian Y, Hoag J, Elliott G, Kukreja R
J Cardiovasc Pharmacol
. 1998 Sep;
32(3):337-42.
PMID: 9733344
We sought to determine the role of opening of adenosine triphosphate (ATP)-sensitive potassium channel (K(ATP) channel) in monophosphoryl lipid A (MLA)-induced myocardial protection after ischemia/reperfusion (I/R) in rabbit. We used...
10.
Szilvassy Z, Ferdinandy P, Cluff C, Elliott G
J Cardiovasc Pharmacol
. 1998 Aug;
32(2):206-12.
PMID: 9700981
We studied whether monophosphoryl lipid A (MLA), an endotoxin derivative, protected the heart from planned ischemia in hypercholesterolemic conscious rabbits. Normal and hypercholesterolemic (8-week exposure to 1.5% cholesterol-enriched diet) conscious...