Francoise Praddaude
Overview
Explore the profile of Francoise Praddaude including associated specialties, affiliations and a list of published articles.
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10
Citations
123
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Recent Articles
1.
Chaabane W, Praddaude F, Buleon M, Jaafar A, Vallet M, Rischmann P, et al.
Am J Physiol Renal Physiol
. 2012 Dec;
304(4):F432-9.
PMID: 23220725
Murine unilateral ureteral obstruction (UUO), a major model of progressive kidney disease, causes loss of proximal tubular mass and formation of atubular glomeruli. Adult C57BL/6 mice underwent a sham operation...
2.
Dupuy V, Mayeur N, Buleon M, Jaafar A, Al Saati T, Schaak S, et al.
Shock
. 2012 Jul;
38(4):351-5.
PMID: 22814286
The objectives of this study were to determine whether type 2 diabetic mice would exhibit a more severe renal impact of hemorrhagic shock (HS) based on a recently described model...
3.
Blaes N, Pecher C, Mehrenberger M, Cellier E, Praddaude F, Chevalier J, et al.
Am J Physiol Renal Physiol
. 2012 May;
303(2):F293-303.
PMID: 22573379
Mesangial matrix expansion is an early lesion leading to glomeruloclerosis and chronic renal diseases. A beneficial effect is achieved with angiotensin I-converting enzyme inhibitors (ACEI), which also favor bradykinin (BK)...
4.
Praddaude F, Cousins S, Pecher C, Marin-Castano M
Exp Eye Res
. 2009 Mar;
89(1):109-18.
PMID: 19281810
Accumulation of specific deposits and extracellular molecules under the retinal pigment epithelium (RPE) has been previously observed in eyes with age-related macular degeneration (AMD) and may play a role in...
5.
Buleon M, Allard J, Jaafar A, Praddaude F, Dickson Z, Ranera M, et al.
Am J Physiol Renal Physiol
. 2008 Mar;
294(5):F1249-56.
PMID: 18367657
Diabetic nephropathy (DN) can be delayed by the use of angiotensin-converting enzyme inhibitors (ACEi). The mechanisms of ACEi renal protection are not univocal. To investigate the impact of bradykinin B(2)...
6.
Buleon M, Mehrenberger M, Pecher C, Praddaude F, Couture R, Tack I, et al.
Med Sci (Paris)
. 2007 Dec;
23(12):1141-7.
PMID: 18154717
Various diseases such as arterial hypertension, diabetes and obesity result in renal diseases which are often irreversible and resistant to currently available therapies. Beside the control of glycemia in diabetic...
7.
Allard J, Buleon M, Cellier E, Renaud I, Pecher C, Praddaude F, et al.
Am J Physiol Renal Physiol
. 2007 Jun;
293(4):F1083-92.
PMID: 17596523
Diabetic nephropathy (DN) is associated with increased oxidative stress, overexpression and activation of growth factor receptors, including those for transforming growth factor-beta1 (TGF-beta-RII), platelet-derived growth factor (PDGF-R), and insulin-like growth...
8.
Schanstra J, Duchene J, Praddaude F, Bruneval P, Tack I, Chevalier J, et al.
Am J Physiol Heart Circ Physiol
. 2003 Feb;
284(6):H1904-8.
PMID: 12560214
Bradykinin B(2) receptor knockout mice (B(2)-/-) have been useful to study the role of bradykinin under pathological conditions. With the use of these mice, it was shown that bradykinin plays...
9.
Marin-Castano M, Schanstra J, Neau E, Praddaude F, Pecher C, Girolami J, et al.
Circulation
. 2002 Feb;
105(5):627-32.
PMID: 11827930
Background: The physiological effects of ACE inhibitors may act in part through a kinin-dependent mechanism. We investigated the effect of chronic ACE-inhibitor treatment on functional kinin B(1)- and B(2)-receptor expression,...
10.
Schanstra J, Marin-Castano M, Praddaude F, Tack I, Girolami J, Bascands J, et al.
J Am Soc Nephrol
. 2000 Jun;
11(7):1208-1215.
PMID: 10864576
Kinins have been shown to influence renal hemodynamics and function. Under physiologic conditions, most kinin effects involve bradykinin B(2) receptors, but bradykinin B(1) receptors are often induced during inflammation. The...