Francisco Fernandez-Belda
Overview
Explore the profile of Francisco Fernandez-Belda including associated specialties, affiliations and a list of published articles.
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Articles
16
Citations
114
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0
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Recent Articles
1.
Asensio-Lopez M, Soler F, Pascual-Figal D, Fernandez-Belda F, Lax A
PLoS One
. 2017 Mar;
12(2):e0172803.
PMID: 28245258
The primary cardiotoxic action of doxorubicin when used as antitumor drug is attributed to the generation of reactive oxygen species (ROS) therefore effective cardioprotection therapies are needed. In this sense,...
2.
Asensio-Lopez M, Soler F, Sanchez-Mas J, Pascual-Figal D, Fernandez-Belda F, Lax A
Arch Biochem Biophys
. 2016 Feb;
594:26-36.
PMID: 26906075
In atrial-derived HL-1 cells, ryanodine receptor and Na(+)/Ca(2+)-exchanger were altered early by 5 μM doxorubicin. The observed effects were an increase of cytosolic Ca(2+) at rest, ensuing ryanodine receptor phosphorylation,...
3.
Soler F, Fernandez-Belda F, Perez-Schindler J, Hernandez-Cascales J
Eur J Pharmacol
. 2015 Sep;
765:429-36.
PMID: 26364538
Cyclic nucleotide phosphodiesterase (PDE)3 and PDE4 provide the major PDE activity in cardiac myocytes and shape β1-adrenoceptor-dependent cardiac cAMP signaling but their role in regulating β2-adrenoceptor-mediated responses is less well...
4.
Soler F, Fernandez-Belda F, Perez-Schindler J, Handschin C, Fuente T, Hernandez-Cascales J
Exp Biol Med (Maywood)
. 2014 Nov;
240(9):1205-13.
PMID: 25432985
The important regulator of cardiac function, cAMP, is hydrolyzed by different cyclic nucleotide phosphodiesterases (PDEs), whose expression and activity are not uniform throughout the heart. Of these enzymes, PDE2 shapes...
5.
Coca R, Soler F, Cortes-Castell E, Gil-Guillen V, Fernandez-Belda F
PLoS One
. 2014 Jul;
9(7):e102083.
PMID: 25003576
Dimethyl-celecoxib is a celecoxib analog that lacks the capacity as cyclo-oxygenase-2 inhibitor and therefore the life-threatening effects but retains the antineoplastic properties. The action mechanism at the molecular level is...
6.
Soler F, Asensio M, Fernandez-Belda F
J Bioenerg Biomembr
. 2012 Aug;
44(5):597-605.
PMID: 22851007
The use of a microsomal preparation from skeletal muscle revealed that both Ca(2+) transport and Ca(2+)-dependent ATP hydrolysis linked to Sarco-Endoplasmic Reticulum Ca(2+)-ATPase are inhibited by epigallocatechin-3-gallate (EGCG). A half-maximal...
7.
Soler F, Lax A, Asensio M, Pascual-Figal D, Fernandez-Belda F
Arch Biochem Biophys
. 2011 Jun;
512(2):175-82.
PMID: 21683055
Increase of resting Ca(2+) levels and amplitude of vasopressin-induced Ca(2+) transients were observed when cells in serum-free medium were exposed to 5mM Ca(2+) for 2h. Small effect on cell viability...
8.
Lax A, Soler F, Fernandez-Belda F
J Bioenerg Biomembr
. 2009 Sep;
41(4):369-77.
PMID: 19777329
The release of cytochrome c from mitochondria induced by 10 microM thapsigargin was linked to rapid loss of the mitochondrial membrane potential whereas that induced by 50 nM staurosporine was...
9.
Coca R, Soler F, Fernandez-Belda F
Arch Biochem Biophys
. 2008 Aug;
478(1):36-42.
PMID: 18675777
The effect of palytoxin was studied in a microsomal fraction enriched in longitudinal tubules of the sarcoplasmic reticulum membrane. Half-maximal effect of palytoxin on Ca(2+)-ATPase activity yielded an apparent inhibition...
10.
Soler F, Lax A, Fernandez-Belda F
Arch Biochem Biophys
. 2007 Jul;
466(2):194-202.
PMID: 17655822
Experimental sarcoplasmic reticulum damage induced by 3 microM thapsigargin or 1 microg/ml tunicamycin provoked viability loss of the cell population in approximately 72 h. Release of cytochrome c from mitochondria...