Eric Vazquez-Ferrer
Overview
Explore the profile of Eric Vazquez-Ferrer including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
7
Citations
543
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Hishida T, Vazquez-Ferrer E, Hishida-Nozaki Y, Takemoto Y, Hatanaka F, Yoshida K, et al.
Front Cell Dev Biol
. 2022 Mar;
10:786031.
PMID: 35309931
It is widely believed that cellular senescence plays a critical role in both aging and cancer, and that senescence is a fundamental, permanent growth arrest that somatic cells cannot avoid....
2.
Huang T, Wang Y, Vazquez-Ferrer E, Theofel I, Requena C, Hanna C, et al.
Nature
. 2021 Dec;
600(7890):737-742.
PMID: 34880491
Stability of the epigenetic landscape underpins maintenance of the cell-type-specific transcriptional profile. As one of the main repressive epigenetic systems, DNA methylation has been shown to be important for long-term...
3.
Hishida T, Vazquez-Ferrer E, Hishida-Nozaki Y, Sancho-Martinez I, Takahashi Y, Hatanaka F, et al.
Protein Cell
. 2019 May;
10(7):485-495.
PMID: 31041783
Identification of the precise molecular pathways involved in oncogene-induced transformation may help us gain a better understanding of tumor initiation and promotion. Here, we demonstrate that SOX2 foregut epithelial cells...
4.
Ocampo A, Reddy P, Martinez-Redondo P, Platero-Luengo A, Hatanaka F, Hishida T, et al.
Cell
. 2016 Dec;
167(7):1719-1733.e12.
PMID: 27984723
Aging is the major risk factor for many human diseases. In vitro studies have demonstrated that cellular reprogramming to pluripotency reverses cellular age, but alteration of the aging process through...
5.
Sancho-Martinez I, Nivet E, Xia Y, Hishida T, Aguirre A, Ocampo A, et al.
Nat Commun
. 2016 Feb;
7:10743.
PMID: 26899176
Glioma tumour-initiating cells (GTICs) can originate upon the transformation of neural progenitor cells (NPCs). Studies on GTICs have focused on primary tumours from which GTICs could be isolated and the...
6.
Aguirre A, Montserrat N, Zacchigna S, Nivet E, Hishida T, Krause M, et al.
Cell Stem Cell
. 2014 Dec;
15(5):589-604.
PMID: 25517466
Heart failure is a leading cause of mortality and morbidity in the developed world, partly because mammals lack the ability to regenerate heart tissue. Whether this is due to evolutionary...
7.
Functional compensation between Myc and PI3K signaling supports self-renewal of embryonic stem cells
Hishida T, Nakachi Y, Mizuno Y, Katano M, Okazaki Y, Ema M, et al.
Stem Cells
. 2014 Nov;
33(3):713-25.
PMID: 25385436
c-Myc and phosphatidylinositol 3-OH kinase (PI3K) both participate in diverse cellular processes, including cell cycle control and tumorigenic transformation. They also contribute to preserving embryonic stem cell (ESC) characteristics. However,...