Elisabetta Trabetti
Overview
Explore the profile of Elisabetta Trabetti including associated specialties, affiliations and a list of published articles.
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64
Citations
2911
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Recent Articles
11.
Fochi S, Lorenzi P, Galasso M, Stefani C, Trabetti E, Zipeto D, et al.
Genes (Basel)
. 2020 Apr;
11(4).
PMID: 32276354
Alternative splicing is a regulatory mechanism essential for cell differentiation and tissue organization. More than 90% of human genes are regulated by alternative splicing events, which participate in cell fate...
12.
Fochi S, Ciminale V, Trabetti E, Bertazzoni U, DAgostino D, Zipeto D, et al.
Pathogens
. 2019 Dec;
8(4).
PMID: 31835460
The risk of developing adult T-cell leukemia/lymphoma (ATLL) in individuals infected with human T-cell lymphotropic virus 1 (HTLV-1) is about 3-5%. The mechanisms by which the virus triggers this aggressive...
13.
Muglia P, Filosi M, Da Ros L, Kam-Thong T, Nardocci F, Trabetti E, et al.
BMC Psychiatry
. 2018 Nov;
18(1):369.
PMID: 30463616
Background: A substantial genetic component accounts for Autism Spectrum Disorders (ASD) aetiology, with some rare and common genetic risk factors recently identified. Large collections of DNAs from thoroughly characterized ASD...
14.
Ligthart S, Vaez A, Vosa U, Stathopoulou M, de Vries P, Prins B, et al.
Am J Hum Genet
. 2018 Nov;
103(5):691-706.
PMID: 30388399
C-reactive protein (CRP) is a sensitive biomarker of chronic low-grade inflammation and is associated with multiple complex diseases. The genetic determinants of chronic inflammation remain largely unknown, and the causal...
15.
Zusi C, Trombetta M, Bonetti S, Dauriz M, Boselli M, Trabetti E, et al.
Diabetes Res Clin Pract
. 2018 Aug;
144:137-143.
PMID: 30153470
This study aims to investigate whether renal and cardiovascular phenotypes in Italian patients with type 2 diabetes (T2D) could be influenced by a number of disease risk SNPs recently found...
16.
Chiarini A, Onorati F, Marconi M, Pasquali A, Patuzzo C, Malashicheva A, et al.
Eur J Prev Cardiol
. 2018 May;
25(1_suppl):51-58.
PMID: 29708036
Background Sporadic non-syndromic thoracic aortic aneurysms (SNSTAAs) are less well understood than familial non-syndromic or syndromic ones. Here, we focused on morphologic and molecular changes of the extracellular matrix of...
17.
Chiarini A, Onorati F, Marconi M, Pasquali A, Patuzzo C, Malashicheva A, et al.
Eur J Prev Cardiol
. 2018 May;
25(1_suppl):42-50.
PMID: 29708032
Background Sporadic non-syndromic thoracic aortic aneurysms (SNSTAAs) are less well understood than familial non-syndromic or syndromic ones. The study aimed at defining the peculiar morphologic and molecular changes occurring in...
18.
Surendran P, Drenos F, Young R, Warren H, Cook J, Manning A, et al.
Nat Genet
. 2016 Sep;
48(10):1151-1161.
PMID: 27618447
High blood pressure is a major risk factor for cardiovascular disease and premature death. However, there is limited knowledge on specific causal genes and pathways. To better understand the genetics...
19.
Torrico B, Chiocchetti A, Bacchelli E, Trabetti E, Hervas A, Franke B, et al.
Autism Res
. 2016 Jul;
10(2):202-211.
PMID: 27417655
Common variants contribute significantly to the genetics of autism spectrum disorder (ASD), although the identification of individual risk polymorphisms remains still elusive due to their small effect sizes and limited...
20.
Huang J, Howie B, McCarthy S, Memari Y, Walter K, Min J, et al.
Nat Commun
. 2015 Sep;
6:8111.
PMID: 26368830
Imputing genotypes from reference panels created by whole-genome sequencing (WGS) provides a cost-effective strategy for augmenting the single-nucleotide polymorphism (SNP) content of genome-wide arrays. The UK10K Cohorts project has generated...