Edward C Carlson
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Explore the profile of Edward C Carlson including associated specialties, affiliations and a list of published articles.
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22
Citations
615
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Recent Articles
1.
Wang K, Dai X, He J, Yan X, Yang C, Fan X, et al.
Diabetes
. 2020 May;
69(8):1779-1792.
PMID: 32404351
Diabetes-induced oxidative stress is one of the major contributors to dysfunction of endothelial progenitor cells (EPCs) and impaired endothelial regeneration. Thus, we tested whether increasing antioxidant protein metallothionein (MT) in...
2.
Carlson E, Chhoun J, Grove B, Laturnus D, Zheng S, Epstein P, et al.
Anat Rec (Hoboken)
. 2016 Nov;
300(3):560-576.
PMID: 27813325
We previously demonstrated that OVE transgenic diabetic mice are susceptible to chronic complications of diabetic nephropathy (DN) including substantial oxidative damage to the renal glomerular filtration barrier (GFB). Importantly, the...
3.
Xu J, Zheng S, Kralik P, Krishnan L, Huang H, Hoying J, et al.
Int J Biol Sci
. 2016 Feb;
12(2):210-8.
PMID: 26884718
The effect of diabetes in vivo has not been examined on isolated podocytes. To achieve this, GFP was expressed constitutively in podocytes of PGFP transgenic mice which were bred to...
4.
Powell D, Kenagy D, Zheng S, Coventry S, Xu J, Cai L, et al.
Life Sci
. 2013 Jun;
93(7):257-64.
PMID: 23800643
Type 1 and Type 2 diabetic patients are at high risk of developing diabetic nephropathy (DN). Renal functional decline is gradual and there is high variability between patients, though the...
5.
Carlson E, Chhoun J, Laturnus D, Bikash K, Berg B, Zheng S, et al.
Diabetes Metab Res Rev
. 2012 Aug;
29(2):113-24.
PMID: 22926941
Background: We previously demonstrated that cellular and extracellular components of the blood-urine barrier in renal glomeruli are susceptible to damage in OVE transgenic mice, a valuable model of human diabetic...
6.
Teiken J, Epstein P, Carlson E
Am J Nephrol
. 2011 Jun;
33 Suppl 1:8-14.
PMID: 21659729
Background/aims: Glomerular lesions in diabetic nephropathy (DN) have been studied in numerous murine diabetic models, but the critical feature of aging is often absent. Since histopathology indicates that in mice,...
7.
Greenwood M, Ressler M, Audette J, Laturnus D, Chhoun R, Teiken J, et al.
Ultrastruct Pathol
. 2011 Feb;
35(2):97-105.
PMID: 21299351
The diabetic-prone BioBreeding Wistar (BB/DP) rat is an autoimmune model of insulin-dependent diabetes mellitus. Approximately 90% of the animals (BB/DPh) are hyperglycemic by 90-120 days of age, while the remaining...
8.
Ceylan-Isik A, Guo K, Carlson E, Privratsky J, Liao S, Cai L, et al.
Hypertension
. 2009 Apr;
53(6):1023-31.
PMID: 19398661
One key mechanism for endothelial dysfunction is endothelial NO synthase (eNOS) uncoupling, whereby eNOS generates O(2)(*-) rather than NO because of deficient eNOS cofactor tetrahydrobiopterin (BH4). This study was designed...
9.
Fogelgren B, Yang S, Sharp I, Huckstep O, Ma W, Somponpun S, et al.
Am J Physiol Renal Physiol
. 2009 Feb;
296(5):F1166-78.
PMID: 19193724
The Br/+ mutant mouse displays decreased embryological expression of the homeobox transcription factor Six2, resulting in hertitable renal hypoplasia. The purpose of this study was to characterize the renal physiological...
10.
Zheng S, Carlson E, Yang L, Kralik P, Huang Y, Epstein P
J Am Soc Nephrol
. 2008 Jul;
19(11):2077-85.
PMID: 18632844
Podocytes are critical components of the selective filtration barrier of the glomerulus and are susceptible to oxidative damage. For investigation of the role of oxidative stress and podocyte damage in...