Douglas Vaughan
Overview
Explore the profile of Douglas Vaughan including associated specialties, affiliations and a list of published articles.
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Snapshot
Articles
7
Citations
237
Followers
0
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Recent Articles
1.
Budinger G, Kohanski R, Gan W, Kobor M, Amaral L, Armanios M, et al.
J Gerontol A Biol Sci Med Sci
. 2017 May;
72(11):1492-1500.
PMID: 28498894
Death from chronic lung disease is increasing and chronic obstructive pulmonary disease has become the third leading cause of death in the United States in the past decade. Both chronic...
2.
Flevaris P, Vaughan D
Semin Thromb Hemost
. 2016 Aug;
43(2):169-177.
PMID: 27556351
Extracellular matrix (ECM) deposition during wound healing is a physiological response to an insult. Wound healing becomes deregulated in the setting of chronic injury or long-standing metabolic disease, leading to...
3.
Davidson L, Wilcox J, Kim D, Benton S, Fredi J, Vaughan D
Am J Med
. 2013 Dec;
127(2):140-4.
PMID: 24332726
Background: Acute coronary syndrome due to acute plaque rupture has been well described and is associated with established risk factors, including hypertension, diabetes mellitus, hyperlipidemia, and smoking. The prevalence of...
4.
Murrow J, Esteves F, Galt J, Chen J, Garcia E, Lin J, et al.
J Nucl Cardiol
. 2011 Jul;
18(5):912-9.
PMID: 21717276
Background: Left ventricular dyssynchrony is an adverse consequence of ST-elevation myocardial infarction (STEMI) and bears an unfavorable prognosis. Mechanical dyssynchrony as measured by phase analysis from gated single photon emission...
5.
Quyyumi A, Waller E, Murrow J, Esteves F, Galt J, Oshinski J, et al.
Am Heart J
. 2010 Dec;
161(1):98-105.
PMID: 21167340
Background: the objective of the study was to determine whether the effects of infarct-related artery (IRA) infusion of autologous bone marrow-derived CD34(+) cells after ST elevation myocardial infarction (STEMI) are...
6.
Salloum J, Tharpe C, Vaughan D, Zhao D
J Invasive Cardiol
. 2005 Nov;
17(11):575-9.
PMID: 16264199
Background: The presence of no-reflow substantially increases the risk of major adverse clinical events (MACE) in percutaneous coronary artery interventions (PCI). Distal protection devices may eliminate both debris and soluble...
7.
Dzau V, Bernstein K, Celermajer D, Cohen J, Dahlof B, Deanfield J, et al.
Cardiovasc Drugs Ther
. 2002 Jul;
16(2):149-60.
PMID: 12090908
Angiotensin-converting enzyme (ACE) activation and the de novo production of angiotensin II contribute to cardiovascular disease through direct pathological tissue effects, including vascular remodeling and inflammation, as well as indirect...