Dimitrios J Stavropoulos
Overview
Explore the profile of Dimitrios J Stavropoulos including associated specialties, affiliations and a list of published articles.
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5
Citations
270
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Recent Articles
1.
Wener E, Cushing S, Papsin B, Stavropoulos D, Mendoza-Londono R, Quercia N, et al.
Otolaryngol Head Neck Surg
. 2025 Feb;
PMID: 40008839
Objective: To assess the added benefit of newborn genetic screening for GJB2 and SLC26A4 variants in conjunction with newborn hearing screening. Study Design: Retrospective cohort study. Methods: Children with known...
2.
Goh E, Stavropoulos D, Adeli K
J Appl Lab Med
. 2021 Jun;
6(5):1299-1304.
PMID: 34086894
Background: Although an obvious critical value in metabolic genetics would be ammonia, it is more challenging to define critical values in molecular genetics and cytogenetics. The objective of this study...
3.
Shlien A, Baskin B, Achatz M, Stavropoulos D, Nichols K, Hudgins L, et al.
Am J Hum Genet
. 2010 Nov;
87(5):631-42.
PMID: 21056402
DNA copy-number variations (CNVs) underlie many neuropsychiatric conditions, but they have been less studied in cancer. We report the association of a 17p13.1 CNV, childhood-onset developmental delay (DD), and cancer....
4.
Pasic I, Shlien A, Durbin A, Stavropoulos D, Baskin B, Ray P, et al.
Cancer Res
. 2010 Jan;
70(1):160-71.
PMID: 20048075
Osteosarcomas are copy number alteration (CNA)-rich malignant bone tumors. Using microarrays, fluorescence in situ hybridization, and quantitative PCR, we characterize a focal region of chr3q13.31 (osteo3q13.31) harboring CNAs in 80%...
5.
Bradshaw P, Stavropoulos D, Meyn M
Nat Genet
. 2005 Jan;
37(2):193-7.
PMID: 15665826
DNA damage surveillance networks in human cells can activate DNA repair, cell cycle checkpoints and apoptosis in response to fewer than four double-strand breaks (DSBs) per genome. These same networks...
6.
Stavropoulos D, Bradshaw P, Li X, Pasic I, Truong K, Ikura M, et al.
Hum Mol Genet
. 2002 Nov;
11(25):3135-44.
PMID: 12444098
Telomerase-negative immortalized human cells maintain telomeres by alternative lengthening of telomeres (ALT) pathway(s), which may involve homologous recombination. We find that endogenous BLM protein co-localizes with telomeric foci in ALT...