Deeann Wallis
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Explore the profile of Deeann Wallis including associated specialties, affiliations and a list of published articles.
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32
Citations
949
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Recent Articles
11.
Carnes R, Kesterson R, Korf B, Mobley J, Wallis D
Genes (Basel)
. 2019 Aug;
10(9).
PMID: 31466283
Neurofibromatosis Type 1 (NF1) is caused by pathogenic variants in the gene encoding neurofibromin. Definition of NF1 protein-protein interactions (PPIs) has been difficult and lacks replication, making it challenging to...
12.
Carnes R, Mobley J, Crossman D, Liu H, Korf B, Kesterson R, et al.
Proteomics
. 2019 Mar;
19(11):e1800334.
PMID: 30908848
Loss of NF1 is an oncogenic driver. In efforts to define pathways responsible for the development of neurofibromas and other cancers, transcriptomic and proteomic changes are evaluated in a non-malignant...
13.
Arcos-Burgos M, Velez J, Martinez A, Ribases M, Ramos-Quiroga J, Sanchez-Mora C, et al.
Transl Psychiatry
. 2019 Jan;
9(1):42.
PMID: 30696812
Genetic factors are strongly implicated in the susceptibility to develop externalizing syndromes such as attention-deficit/hyperactivity disorder (ADHD), oppositional defiant disorder, conduct disorder, and substance use disorder (SUD). Variants in the...
14.
Isakov O, Wallis D, Evans D, Ben-Shachar S
EBioMedicine
. 2018 Oct;
36:508-516.
PMID: 30274822
Background: Neurofibromatosis type I (NF1) is caused by heterozygous loss-of-function variants in the NF1 gene encoding neurofibromin which serves as a tumor suppressor that inhibits RAS signaling and regulates cell...
15.
Wallis D, Li K, Lui H, Hu K, Chen M, Li J, et al.
Hum Mutat
. 2018 Mar;
39(6):816-821.
PMID: 29522274
Neurofibromatosis type 1 (NF1) is caused by pathogenic variants or mutations in the NF1 gene that encodes neurofibromin. We describe here a new approach to determining the functional consequences of...
16.
Aggarwal A, Parai M, Shetty N, Wallis D, Woolhiser L, Hastings C, et al.
Cell
. 2017 Jul;
170(2):249-259.e25.
PMID: 28669536
Widespread resistance to first-line TB drugs is a major problem that will likely only be resolved through the development of new drugs with novel mechanisms of action. We have used...
17.
Orsini C, Setlow B, DeJesus M, Galaviz S, Loesch K, Ioerger T, et al.
Mol Genet Genomic Med
. 2016 Jun;
4(3):322-43.
PMID: 27247960
Background: The Latrophilin 3 (LPHN3) gene (recently renamed Adhesion G protein-coupled receptor L3 (ADGRL3)) has been linked to susceptibility to attention deficit/hyperactivity disorder (ADHD) and vulnerability to addiction. However, its...
18.
Larroquette F, Seto L, Gaub P, Kamal B, Wallis D, Lariviere R, et al.
Hum Mol Genet
. 2015 Sep;
24(22):6515-29.
PMID: 26362257
Missense mutations (P56S) in Vapb are associated with autosomal dominant motor neuron diseases: amyotrophic lateral sclerosis and lower motor neuron disease. Although transgenic mice overexpressing the mutant vesicle-associated membrane protein-associated...
19.
Loesch K, Galaviz S, Hamoui Z, Clanton R, Akabani G, Deveau M, et al.
PLoS One
. 2015 Apr;
10(4):e0120534.
PMID: 25853515
Elucidating the genetic determinants of radiation response is crucial to optimizing and individualizing radiotherapy for cancer patients. In order to identify genes that are involved in enhanced sensitivity or resistance...
20.
Wallis D, Loesch K, Galaviz S, Sun Q, DeJesus M, Ioerger T, et al.
Stem Cells
. 2015 Mar;
33(8):2509-22.
PMID: 25752821
We used a genomic library of mutant murine embryonic stem cells (ESCs) and report the methodology required to simultaneously culture, differentiate, and screen more than 3,200 heterozygous mutant clones to...