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David E Theobald

Explore the profile of David E Theobald including associated specialties, affiliations and a list of published articles. Areas
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Articles 16
Citations 1159
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Recent Articles
11.
Tait D, Brown V, Farovik A, Theobald D, Dalley J, Robbins T
Eur J Neurosci . 2007 Jul; 25(12):3719-24. PMID: 17610591
Rats with medial prefrontal cortex (mPFC) lesions are impaired in attentional set-shifting, when it is required to shift to a previously irrelevant perceptual dimension. The main source of noradrenergic input...
12.
Russo S, Bolanos C, Theobald D, DeCarolis N, Renthal W, Kumar A, et al.
Nat Neurosci . 2006 Dec; 10(1):93-9. PMID: 17143271
Chronic morphine administration (via subcutaneous pellet) decreases the size of dopamine neurons in the ventral tegmental area (VTA), a key reward region in the brain, yet the molecular basis and...
13.
Green T, Alibhai I, Hommel J, DiLeone R, Kumar A, Theobald D, et al.
J Neurosci . 2006 Aug; 26(32):8235-42. PMID: 16899718
Previous research has shown that cAMP response element (CRE)-mediated transcription is activated in the nucleus accumbens, a major brain reward region, by a variety of environmental stimuli and contributes to...
14.
Dalley J, Theobald D, Bouger P, Chudasama Y, Cardinal R, Robbins T
Cereb Cortex . 2004 Apr; 14(8):922-32. PMID: 15084496
Lesions of the basal forebrain (BF) cortical cholinergic system impair performance on a rodent five-choice visual attentional task. This study examines the effects on the same task of selective depletion...
15.
Dalley J, Chudasama Y, Theobald D, Pettifer C, Fletcher C, Robbins T
Psychopharmacology (Berl) . 2002 Jun; 161(4):425-33. PMID: 12073171
Rationale: Although dopaminergic mechanisms have been implicated in incentive motivational processes, their role in appetitive conditioning remains poorly understood. Objectives: To investigate the effects of dopamine (DA) depleting lesions of...
16.
Dalley J, Theobald D, Eagle D, Passetti F, Robbins T
Neuropsychopharmacology . 2002 May; 26(6):716-28. PMID: 12007742
Converging lines of evidence suggest that dysfunction of brain serotonergic systems may underlie impulsive behavior. However, the nature of this deficit remains poorly understood because indirect indices of serotonin (5-HT)...