Christian Deuschle
Overview
Explore the profile of Christian Deuschle including associated specialties, affiliations and a list of published articles.
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45
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1113
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Recent Articles
1.
Zimmermann M, Fandrich M, Jakobi M, Roben B, Wurster I, Lerche S, et al.
J Parkinsons Dis
. 2024 Sep;
14(7):1405-1416.
PMID: 39240648
Background: Prior investigations have elucidated pathophysiological interactions involving blood coagulation and neurodegenerative diseases. These interactions pertain to age-related effects and a mild platelet antiaggregant function of exogenous α-Synuclein. Objective: Our...
2.
Zimmermann M, Fandrich M, Jakobi M, Roben B, Wurster I, Lerche S, et al.
Eur J Neurol
. 2024 Jul;
31(10):e16388.
PMID: 38946703
Background And Purpose: Parkinson's disease (PD) is an age-related condition characterized by substantial phenotypic variability. Consequently, pathways and proteins involved in biological aging, such as the central aging pathway comprising...
3.
Schaeffer E, Kluge A, Schulte C, Deuschle C, Bunk J, Welzel J, et al.
J Parkinsons Dis
. 2024 Apr;
14(4):667-679.
PMID: 38669557
Background: Misfolded α-synuclein can be detected in blood samples of Parkinson's disease (PD) patients by a seed amplification assay (SAA), but the association with disease duration is not clear, yet....
4.
Roeben B, Liepelt-Scarfone I, Lerche S, Zimmermann M, Wurster I, Sunkel U, et al.
NPJ Parkinsons Dis
. 2024 Apr;
10(1):88.
PMID: 38649346
With disease-modifying treatment for Parkinson's disease (PD) associated with variants in the glucocerebrosidase gene (GBA1) under way, the challenge to design clinical trials with non-PD-manifest GBA mutation carriers (GBA1) comes...
5.
Brockmann K, Lerche S, Baiardi S, Rossi M, Wurster I, Quadalti C, et al.
NPJ Parkinsons Dis
. 2024 Jan;
10(1):24.
PMID: 38242875
Seed amplification assays have been implemented in Parkinson's disease to reveal disease-specific misfolded alpha-synuclein aggregates in biospecimens. While the assays' qualitative dichotomous seeding response is valuable to stratify and enrich...
6.
Roeben B, Scharf M, Miske R, Teegen B, Traschutz A, Wilke C, et al.
J Neurol
. 2023 Jul;
270(11):5649-5654.
PMID: 37507501
No abstract available.
7.
Lerche S, Zimmermann M, Roeben B, Wurster I, Fries F, Deuschle C, et al.
NPJ Parkinsons Dis
. 2023 Mar;
9(1):38.
PMID: 36906614
Inflammation modifies the incidence and progression of Parkinson's disease (PD). By using 30 inflammatory markers in CSF in 498 people with PD and 67 people with dementia with Lewy bodies...
8.
Wurster I, Quadalti C, Rossi M, Hauser A, Deuschle C, Schulte C, et al.
NPJ Parkinsons Dis
. 2022 Sep;
8(1):117.
PMID: 36109514
Lewy-body pathology with aggregation of abnormal conformations of the protein alpha-synuclein (α-Syn) represent the histopathological hallmarks of Parkinson's disease (PD). Genetic prototypes such as PD due to mutations in the...
9.
Lerche S, Zimmermann M, Wurster I, Roeben B, Fries F, Deuschle C, et al.
Front Neurol
. 2022 Mar;
13:834580.
PMID: 35280273
Background: An involvement of the central-nervous and peripheral, innate and adaptive immune system in the pathogenesis of Parkinson's disease (PD) is nowadays well established. Objectives: We face several open questions...
10.
Brockmann K, Quadalti C, Lerche S, Rossi M, Wurster I, Baiardi S, et al.
Acta Neuropathol Commun
. 2021 Oct;
9(1):175.
PMID: 34717775
The clinicopathological heterogeneity in Lewy-body diseases (LBD) highlights the need for pathology-driven biomarkers in-vivo. Misfolded alpha-synuclein (α-Syn) is a lead candidate based on its crucial role in disease pathophysiology. Real-time...