Charlotte Israelsson
Overview
Explore the profile of Charlotte Israelsson including associated specialties, affiliations and a list of published articles.
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11
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285
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Recent Articles
1.
Lindblom R, Molnar M, Israelsson C, Rojsater B, Wiklund L, Lennmyr F
J Stroke Cerebrovasc Dis
. 2018 Jan;
27(5):1200-1211.
PMID: 29306595
Background: Survivors of cardiac arrest often experience neurologic deficits. To date, treatment options are limited. Associated hyperglycemia is believed to further worsen the neurologic outcome. The aim with this study...
2.
Israelsson C, Kylberg A, Bjorklund U, Ebendal T
J Neurosci Res
. 2015 Jun;
93(10):1519-25.
PMID: 26068706
A major component of the damaging effect after traumatic brain injury (TBI) is activation of the inflammatory system. In particular, chemokines and chemokine-regulated factors become activated in resident brain cells...
3.
Israelsson C, Kylberg A, Bengtsson H, Hillered L, Ebendal T
PLoS One
. 2014 Aug;
9(8):e104754.
PMID: 25153123
Brain trauma is known to activate inflammatory cells via various chemokine signals although their interactions remain to be characterized. Mice deficient in Ccl3, Ccr2 or Cxcl10 were compared with wildtype...
4.
Israelsson C, Flygt J, Astrand E, Kiwanuka O, Bengtsson H, Marklund N
Restor Neurol Neurosci
. 2014 Aug;
32(5):717-31.
PMID: 25079982
Purpose: When central nervous system axons are injured, regeneration is partly inhibited by myelin-associated inhibitors (MAIs). Following traumatic brain injury (TBI) in the rat, pharmacological neutralisation of the MAIs Nogo-A...
5.
Hanell A, Clausen F, Djupsjo A, Vallstedt A, Patra K, Israelsson C, et al.
J Neurotrauma
. 2012 Sep;
29(17):2660-71.
PMID: 22985250
We investigated the role of the axon guidance molecule EphA4 following traumatic brain injury (TBI) in mice. Neutralization of EphA4 improved motor function and axonal regeneration following experimental spinal cord...
6.
Clausen F, Hanell A, Israelsson C, Hedin J, Ebendal T, Mir A, et al.
Eur J Neurosci
. 2011 Jun;
34(1):110-23.
PMID: 21623956
Increasing evidence suggests that interleukin-1β (IL-1β) is a key mediator of the inflammatory response following traumatic brain injury (TBI). Recently, we showed that intracerebroventricular administration of an IL-1β-neutralizing antibody was...
7.
Ekmark-Lewen S, Lewen A, Israelsson C, Li G, Farooque M, Olsson Y, et al.
Restor Neurol Neurosci
. 2010 May;
28(3):311-21.
PMID: 20479526
Purpose: Astroglial responses after traumatic brain injury are difficult to detect with routine morphological methods. The aims for this study were to compare the temporal and spatial expression pattern of...
8.
Israelsson C, Bengtsson H, Lobell A, Nilsson L, Kylberg A, Isaksson M, et al.
Eur J Neurosci
. 2010 Apr;
31(5):852-63.
PMID: 20374285
Traumatic brain injury (TBI) in the mouse results in the rapid appearance of scattered clusters of cells expressing the chemokine Cxcl10 in cortical and subcortical areas. To extend the observation...
9.
Israelsson C, Wang Y, Kylberg A, Pick C, Hoffer B, Ebendal T
J Neurotrauma
. 2009 Mar;
26(8):1307-14.
PMID: 19317611
Cerebral gene expression changes in response to traumatic brain injury will provide useful information in the search for future trauma treatment. In order to characterize the outcome of mild brain...
10.
Israelsson C, Bengtsson H, Kylberg A, Kullander K, Lewen A, Hillered L, et al.
J Neurotrauma
. 2008 Jul;
25(8):959-74.
PMID: 18665806
Cerebral gene expressions change in response to traumatic brain injury (TBI), and future trauma treatment may improve with increased knowledge about these regulations. We subjected C57BL/6J mice to injury by...