Cees J Tack
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Explore the profile of Cees J Tack including associated specialties, affiliations and a list of published articles.
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180
Citations
4204
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Recent Articles
1.
Fabricius T, Verhulst C, Svensson C, Wienberg M, Duijnhouwer A, Tack C, et al.
Diabetes Obes Metab
. 2025 Mar;
PMID: 40045554
Aims: Cardiovascular disease is the most common complication and cause of death in people with diabetes. Hypoglycaemia is independently associated with the development of cardiovascular complications, including death. The aim...
2.
Tans R, Glendorf T, van Herwaarden A, Venselaar H, van Rijswijck D, Wevers R, et al.
BMJ Open Diabetes Res Care
. 2024 Dec;
12(6.
PMID: 39706672
Introduction: Maturity-onset diabetes of the young (MODY) and neonatal diabetes mellitus (NDM) are the most prevalent causes of monogenic diabetes. MODY is an autosomal dominant condition with onset in childhood...
3.
van Heck J, Ajie M, Joosten L, Tack C, Stienstra R
Diabetes Obes Metab
. 2024 Nov;
27(2):719-728.
PMID: 39562286
Background: The presence of low-grade inflammation has been reported in people with type 2 diabetes and related to the development of (macro)vascular complications. Whether systemic inflammation is present in type...
4.
Tokgoz S, Boss M, Jansen T, Jansen T, Meijer R, Frielink C, et al.
Diabetes
. 2024 Nov;
74(2):212-222.
PMID: 39561332
It is unclear why some individuals with type 2 diabetes are unresponsive to treatment with glucagon-like peptide 1 receptor (GLP-1R) agonists, but hypothalamic-pituitary-adrenal (HPA) axis activation could play a role....
5.
He X, Wang X, van Heck J, van Cranenbroek B, van Rijssen E, Stienstra R, et al.
Front Immunol
. 2024 Jul;
15:1401542.
PMID: 39011037
Aims/hypothesis: There is increasing evidence for heterogeneity in type 1 diabetes mellitus (T1D): not only the age of onset and disease progression rate differ, but also the risk of complications...
6.
Ajie M, van Heck J, Verhulst C, Fabricius T, Hendriksz M, McCrimmon R, et al.
Diabetes Obes Metab
. 2024 Jun;
26(9):3696-3704.
PMID: 38899554
Aim: To determine whether recent repeated exposure to real-life hypoglycaemia affects the pro-inflammatory response during a hypoglycemia episode. Materials And Methods: This was a post hoc analysis of a hyperinsulinaemic...
7.
Svensson C, Fabricius T, Verhulst C, Kristensen P, Tack C, Heller S, et al.
Diabetes Obes Metab
. 2024 May;
26(8):3213-3222.
PMID: 38774963
Aim: Experimental hypoglycaemia blunts the counterregulatory hormone and symptom responses to a subsequent episode of hypoglycaemia. In this study, we aimed to assess the associations between antecedent exposure and continuous...
8.
Drenthen L, Ajie M, de Baaij J, Tack C, de Galan B, Stienstra R
J Clin Endocrinol Metab
. 2024 Feb;
109(12):e2240-e2245.
PMID: 38412117
Context: Low magnesium levels, which are common in people with type 2 diabetes, are associated with increased levels of proinflammatory molecules. It is unknown whether magnesium supplementation decreases this low-grade...
9.
Fabricius T, Verhulst C, Kristensen P, Holst J, Tack C, McCrimmon R, et al.
Acta Diabetol
. 2024 Feb;
61(5):623-633.
PMID: 38376580
Aim: The sympathetic nervous and hormonal counterregulatory responses to hypoglycaemia differ between people with type 1 and type 2 diabetes and may change along the course of diabetes, but have...
10.
Verhulst C, van Heck J, Fabricius T, Stienstra R, Teerenstra S, McCrimmon R, et al.
Cardiovasc Diabetol
. 2024 Feb;
23(1):55.
PMID: 38331900
Background: Hypoglycaemia has been shown to induce a systemic pro-inflammatory response, which may be driven, in part, by the adrenaline response. Prior exposure to hypoglycaemia attenuates counterregulatory hormone responses to...