C Lucchinetti
Overview
Explore the profile of C Lucchinetti including associated specialties, affiliations and a list of published articles.
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15
Citations
2996
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Recent Articles
1.
Pathological heterogeneity of idiopathic central nervous system inflammatory demyelinating disorders
Lucchinetti C
Curr Top Microbiol Immunol
. 2008 Jan;
318:19-43.
PMID: 18219813
The last decade has seen a resurgence of interest in MS neuropathology. This resurgence was partly fueled by the development of new molecular and histochemical tools to examine the MS...
2.
Schwartz K, Erickson B, Lucchinetti C
Neuroradiology
. 2006 Feb;
48(3):143-9.
PMID: 16447037
Ring-enhancing lesions seen on MR images can occur with a variety of etiologies. Some ring-enhancing lesions have hypointense rims peripherally on T2-weighted MR images. In this study, we examined whether...
3.
Frohman E, Filippi M, Stuve O, Waxman S, Corboy J, Phillips J, et al.
Arch Neurol
. 2005 Sep;
62(9):1345-56.
PMID: 16157741
Major advancements have been achieved in our ability to diagnose multiple sclerosis (MS) and to commence treatment intervention with agents that can favorably affect the disease course. Although MS exacerbations...
4.
Hoftberger R, Aboul-Enein F, Brueck W, Lucchinetti C, Rodriguez M, Schmidbauer M, et al.
Brain Pathol
. 2004 Mar;
14(1):43-50.
PMID: 14997936
Multiple sclerosis is considered to be an immune-mediated disease of the central nervous system, characterized by chronic inflammation, primary demyelination and axonal damage. The mechanisms of demyelination and axonal injury...
5.
Bruck W, Lucchinetti C, Lassmann H
Mult Scler
. 2002 May;
8(2):93-7.
PMID: 11990878
The present review will focus on the current knowledge of the pathology of primary progressive multiple sclerosis lesions. Multiple sclerosis (MS) is an inflammatory demyelinating disease with a broad clinical...
6.
Bitsch A, Kuhlmann T, Stadelmann C, Lassmann H, Lucchinetti C, Bruck W
Ann Neurol
. 2001 Jun;
49(6):793-6.
PMID: 11409432
Severe tissue destruction is the presumed histopathological correlate of hypointense multiple sclerosis (MS) lesions. In this study we correlated changes of lesion hypointensity over time with initial histopathological features in...
7.
Lucchinetti C, Bruck W, Noseworthy J
Curr Opin Neurol
. 2001 May;
14(3):259-69.
PMID: 11371747
The cause of multiple sclerosis is generally considered to be entirely T cell mediated. However, recent reports of studies in a variety of animal models of inflammatory demyelinating disease, coupled...
8.
Lassmann H, Bruck W, Lucchinetti C
Trends Mol Med
. 2001 Apr;
7(3):115-21.
PMID: 11286782
Multiple sclerosis is a chronic inflammatory disease of the nervous system in which a T-cell-mediated inflammatory process is associated with destruction of myelin sheaths. Although demyelination is the primary event,...
9.
Noseworthy J, Lucchinetti C, Rodriguez M, Weinshenker B
N Engl J Med
. 2000 Sep;
343(13):938-52.
PMID: 11006371
No abstract available.
10.
Lucchinetti C, Bruck W, Parisi J, Scheithauer B, Rodriguez M, Lassmann H
Ann Neurol
. 2000 Jun;
47(6):707-17.
PMID: 10852536
Multiple sclerosis (MS) is a disease with profound heterogeneity in clinical course, neuroradiological appearance of the lesions, involvement of susceptibility gene loci, and response to therapy. These features are supported...