C John Sperati
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Explore the profile of C John Sperati including associated specialties, affiliations and a list of published articles.
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65
Citations
704
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Recent Articles
1.
Sun Y, Kronenberg N, Sethi S, Dash S, Kovalik M, Sempowski B, et al.
Am J Physiol Renal Physiol
. 2025 Mar;
PMID: 40062402
Focal Segmental Glomerulosclerosis (FSGS) is a histologic lesion caused by a variety of injurious stimuli that lead to dysfunction/loss of glomerular visceral epithelial cells (i.e. podocytes). Pathogenic mutations in CRB2,...
2.
Tamargo C, Sperati C, Zhang C, ORourke Jr P, Sozio S
Am J Kidney Dis
. 2025 Feb;
PMID: 39892460
No abstract available.
3.
Gupta S, Kaunfer S, Chen K, Dias J, Vijayan A, Rajasekaran A, et al.
Blood
. 2025 Jan;
PMID: 39760780
High-dose methotrexate (MTX) results in high rates of acute kidney injury (AKI), neutropenia, and hepatotoxicity. Glucarpidase is a recombinant enzyme that cleaves MTX, but clinical data supporting its use are...
4.
Sun Y, Kronenberg N, Sethi S, Dash S, Kovalik M, Sempowski B, et al.
bioRxiv
. 2024 Nov;
PMID: 39484460
Focal Segmental Glomerulosclerosis (FSGS) is a histologic lesion caused by a variety of injurious stimuli that lead to dysfunction/loss of glomerular visceral epithelial cells (i.e. podocytes). Pathogenic mutations in CRB2,...
5.
Cole M, Ranjan N, Gerber G, Pan X, Flores-Guerrero D, McNamara G, et al.
Blood
. 2024 Oct;
144(24):2528-2545.
PMID: 39357054
Complement-mediated thrombotic microangiopathy (CM-TMA) or hemolytic uremic syndrome, previously identified as atypical hemolytic uremic syndrome, is a TMA characterized by germ line variants or acquired antibodies to complement proteins and...
6.
Hannouneh Z, Cervantes C, Sperati C, Hanouneh M
BMC Nephrol
. 2024 Sep;
25(1):315.
PMID: 39333966
Background: Familial hypokalemic periodic paralysis (HypoPP) is an uncommon genetic disorder characterized by recurrent episodes of muscle weakness and hypokalemia, typically starting in early adulthood. The existence of hyperthyroidism in...
7.
Nester C, Feldman D, Burwick R, Cataland S, Chaturvedi S, Cook H, et al.
Kidney Int
. 2024 Aug;
106(3):326-336.
PMID: 39174192
The term atypical hemolytic uremic syndrome has been in use since the mid-1970s. It was initially used to describe the familial or sporadic form of hemolytic uremic syndrome as opposed...
8.
Fatola A, Johnson B, Walsh L, Fang D, White M, Le D, et al.
BMC Nephrol
. 2024 Jun;
25(1):200.
PMID: 38890600
Malakoplakia is a rare inflammatory disorder believed to result from a defect in macrophage phagocytic function triggering a granulomatous reaction. It can present with genitourinary, gastrointestinal, or cutaneous manifestations in...
9.
Cole M, Ranjan N, Gerber G, Pan X, Flores-Guerrero D, Chaturvedi S, et al.
bioRxiv
. 2024 Jun;
PMID: 38854038
Key Points: CM-HUS has a CP stimulus driven by polyreactive IgM, addressing the mystery of why 40% of CM-HUS lack complement specific variantsComplement biosensors and the bioluminescent mHam can be...
10.
Barratt J, Liew A, Yeo S, Fernstrom A, Barbour S, Sperati C, et al.
Clin J Am Soc Nephrol
. 2024 Jan;
19(4):452-462.
PMID: 38214599
Background: IgA nephropathy is the most common primary GN. Clinical features of IgA nephropathy include proteinuria, which is the strongest known surrogate of progression to kidney failure. Complement pathway activation...