C H Conrad
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Explore the profile of C H Conrad including associated specialties, affiliations and a list of published articles.
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Articles
33
Citations
562
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Recent Articles
1.
Brooks W, BING O, Boluyt M, Malhotra A, Morgan J, Satoh N, et al.
Hypertension
. 2000 Jun;
35(6):1203-9.
PMID: 10856264
Inotropic responsiveness to beta-adrenergic stimulation is generally found to be impaired in left ventricular (LV) hypertrophy and failure. To investigate the mechanisms by which angiotensin-converting enzyme inhibitor therapy may modulate...
2.
Brooks W, Conrad C
J Mol Cell Cardiol
. 2000 Mar;
32(2):187-95.
PMID: 10722796
Aging is associated with an increase in myocardial extracellular matrix components and contractile dysfunction. Transforming growth factor- beta(1)(TGF- beta(1)) has been shown to regulate expression of collagen genes and extracellular...
3.
Brooks W, Conrad C
Comp Biochem Physiol A Mol Integr Physiol
. 2000 Jan;
124(2):139-47.
PMID: 10629955
Genetically altered mice have become an increasingly important tool for the study of mechanisms of cardiac function, and therefore it is vital to characterize the basic contractile properties of the...
4.
Brooks W, Garibaldi B, Conrad C
Lab Anim Sci
. 1999 Mar;
48(4):374-8.
PMID: 10090046
A simplified transthoracic procedure using electrocauterization was used to induce myocardial injury in mice. After a single small incision through the skin and dissection of the underlying musculature, a modified...
5.
Singh K, Sirokman G, Communal C, Robinson K, Conrad C, Brooks W, et al.
Hypertension
. 1999 Feb;
33(2):663-70.
PMID: 10024324
To identify genes that are differentially expressed during the transition from compensated hypertrophy to failure, myocardial mRNA from spontaneously hypertensive rats (SHR) with heart failure (SHR-F) was compared with that...
6.
Zile M, Koide M, Sato H, Ishiguro Y, Conrad C, Buckley J, et al.
J Am Coll Cardiol
. 1999 Feb;
33(1):250-60.
PMID: 9935038
Objectives: We sought to determine whether the ameliorative effects of microtubule depolymerization on cellular contractile dysfunction in pressure overload cardiac hypertrophy apply at the tissue level. Background: A selective and...
7.
Cicogna A, Robinson K, Conrad C, Singh K, Squire R, Okoshi M, et al.
Hypertension
. 1999 Feb;
33(1):60-5.
PMID: 9931082
-The aging spontaneously hypertensive rat (SHR) is a model in which the transition from chronic stable left ventricular hypertrophy to overt heart failure can be observed. Although the mechanisms for...
8.
Cicogna A, Robinson K, Conrad C, Squire R, Okoshi M, BING O
Arq Bras Cardiol
. 1998 Jun;
69(6):381-4.
PMID: 9609008
Purpose: To investigate the participation of contractile state and relaxation in cardiac muscle dysfunction during the transition from stable hypertrophy to cardiac decompensation in aging spontaneously hypertensive rats (SHR). Methods:...
9.
Brooks W, BING O, Robinson K, Slawsky M, Chaletsky D, Conrad C
Circulation
. 1997 Dec;
96(11):4002-10.
PMID: 9403625
Background: After a period of stable hypertrophy, male spontaneously hypertensive rats (SHR) develop heart failure between 18 to 24 months of age, with depression of active myocardial function and increased...
10.
Brooks W, BING O, Conrad C, ONeill L, Crow M, Lakatta E, et al.
Hypertension
. 1997 Dec;
30(6):1362-8.
PMID: 9403554
The spontaneously hypertensive rat (SHR) exhibits a transition from stable compensated left ventricular (LV) hypertrophy to heart failure (HF) at a mean age of 21 months that is characterized by...