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C Godinot

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Articles 107
Citations 444
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Recent Articles
1.
Le Dizes S, Aulagnier C, Maro D, Rozet M, Vermorel F, Hebert D, et al.
J Environ Radioact . 2017 Feb; 171:83-92. PMID: 28199861
In this paper, a dynamic compartment model with a high temporal resolution has been investigated to describe tritium transfer in grassland ecosystems exposed to atmospheric H releases from nuclear facilities...
2.
Maro D, Vermorel F, Rozet M, Aulagnier C, Hebert D, Le Dizes S, et al.
J Environ Radioact . 2016 Dec; 167:235-248. PMID: 27908461
Tritium (H) is mainly released into the environment by nuclear power plants, military nuclear facilities and nuclear reprocessing plants. The construction of new nuclear facilities in the world as well...
3.
Connan O, Solier L, Hebert D, Maro D, Lamotte M, Voiseux C, et al.
J Environ Radioact . 2014 Aug; 137:142-149. PMID: 25078471
The aim of this work was to study the near-field dispersion of (85)Kr around the nuclear fuel reprocessing plant at La Hague (AREVA NC La Hague - France) under stable...
4.
Tavernier L, Godinot C
Lyon Chir . 2010 Mar; 42(1):5-12. PMID: 20247274
No abstract available.
5.
Dubot A, Hervouet E, Mandon G, Zabot M, Godinot C
Mitochondrion . 2005 Aug; 4(1):41-7. PMID: 16120373
Leigh syndrome with cytochrome oxidase (COX) deficiency has been associated with SURF1 mutations. For patient diagnosis, distinction between neutral polymorphisms and pathogenic missense SURF1 mutations in Leigh syndrome is essential....
6.
Dubot A, Godinot C, Dumur V, Sablonniere B, Stojkovic T, Cuisset J, et al.
Biochem Biophys Res Commun . 2003 Dec; 313(3):687-93. PMID: 14697245
A maternally inherited and practically homoplasmic mitochondrial (mtDNA) mutation, 8527A>G, changing the initiation codon AUG into GUG, normally coding for a valine, was observed in the ATP6 gene encoding the...
7.
Capkova M, Hansikova H, Godinot C, Houstkova H, Houstek J, Zeman J
Cas Lek Cesk . 2003 Jan; 141(20):636-41. PMID: 12515039
Background: Leigh disease, subacute necrotizing encephalopathy, is a serious mitochondrial disorder of energy-providing metabolism. Clinical presentation usually starts in infancy as a progressive neurodegenerative disorder with retardation and regression of...
8.
Liu J, Wang C, Murakami Y, Gong G, Ishibashi Y, Prody C, et al.
Am J Physiol Heart Circ Physiol . 2001 Aug; 281(3):H1319-26. PMID: 11514303
This study examined high-energy phosphates (HEP) and mitochondrial ATPase protein expression in hearts in which myocardial infarction resulted in either compensated left ventricular remodeling (LVR) or congestive heart failure (CHF)....
9.
Legros F, Chatzoglou E, Frachon P, Ogier de Baulny H, Laforet P, Jardel C, et al.
Eur J Hum Genet . 2001 Jul; 9(7):510-8. PMID: 11464242
The great variability of the human mitochondrial DNA (mtDNA) sequence induces many difficulties in the search for its deleterious mutations. We illustrate these pitfalls by the analysis of the cytochrome...
10.
Pequignot M, Dey R, Zeviani M, Tiranti V, Godinot C, Poyau A, et al.
Hum Mutat . 2001 Apr; 17(5):374-81. PMID: 11317352
Cytochrome c oxidase (COX) deficiency is one of the major causes of Leigh Syndrome (LS), a fatal encephalopathy of infancy or childhood, characterized by symmetrical lesions in the basal ganglia...