Boris Pantic
Overview
Explore the profile of Boris Pantic including associated specialties, affiliations and a list of published articles.
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Snapshot
Articles
9
Citations
330
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0
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Recent Articles
1.
Pantic B, Ives D, Mennuni M, Perez-Rodriguez D, Fernandez-Pelayo U, de Arbina A, et al.
Nat Commun
. 2021 Dec;
12(1):6997.
PMID: 34873176
Pathological variants of human mitochondrial DNA (mtDNA) typically co-exist with wild-type molecules, but the factors driving the selection of each are not understood. Because mitochondrial fitness does not favour the...
2.
Semplicini C, Bertolin C, Bello L, Pantic B, Guidolin F, Vianello S, et al.
Neurology
. 2018 Sep;
91(17):e1629-e1641.
PMID: 30258016
Objective: To identify and characterize patients with calsequestrin 1 ()-related myopathy. Methods: Patients selected according to histopathologic features underwent genetic screening. mutated patients were clinically evaluated and underwent muscle MRI....
3.
Vianello S, Pantic B, Fusto A, Bello L, Galletta E, Borgia D, et al.
Hum Mol Genet
. 2017 Jun;
26(17):3342-3351.
PMID: 28595270
Glucocorticoids are beneficial in Duchenne muscular dystrophy (DMD). Osteopontin (OPN), the protein product of SPP1, plays a role in DMD pathology modulating muscle inflammation and regeneration. A polymorphism in the...
4.
Malena A, Pantic B, Borgia D, Sgarbi G, Solaini G, Holt I, et al.
Autophagy
. 2016 Sep;
12(11):2098-2112.
PMID: 27627835
Pathological mutations in the mitochondrial DNA (mtDNA) produce a diverse range of tissue-specific diseases and the proportion of mutant mitochondrial DNA can increase or decrease with time via segregation, dependent...
5.
Pantic B, Borgia D, Giunco S, Malena A, Kiyono T, Salvatori S, et al.
Exp Cell Res
. 2016 Feb;
342(1):39-51.
PMID: 26905645
Primary human skeletal muscle cells (hSkMCs) are invaluable tools for deciphering the basic molecular mechanisms of muscle-related biological processes and pathological alterations. Nevertheless, their use is quite restricted due to...
6.
Barp A, Bello L, Politano L, Melacini P, Calore C, Polo A, et al.
PLoS One
. 2015 Oct;
10(10):e0141240.
PMID: 26513582
Objective: Dilated cardiomyopathy (DCM) is a major complication and leading cause of death in Duchenne muscular dystrophy (DMD). DCM onset is variable, suggesting modifier effects of genetic or environmental factors....
7.
Botta A, Malena A, Loro E, Del Moro G, Suman M, Pantic B, et al.
Genes (Basel)
. 2014 Apr;
4(2):275-92.
PMID: 24705164
The pathogenesis of Myotonic Dystrophy type 1 (DM1) is linked to unstable CTG repeats in the DMPK gene which induce the mis-splicing to fetal/neonatal isoforms of many transcripts, including those...
8.
Rasola A, Sciacovelli M, Pantic B, Bernardi P
FEBS Lett
. 2010 Feb;
584(10):1989-96.
PMID: 20153328
The permeability transition pore (PTP) is an inner mitochondrial membrane channel that has been thoroughly characterized functionally, yet remains an elusive molecular entity. The best characterized PTP-regulatory component, cyclophilin (CyP)...
9.
Rasola A, Sciacovelli M, Chiara F, Pantic B, Brusilow W, Bernardi P
Proc Natl Acad Sci U S A
. 2010 Jan;
107(2):726-31.
PMID: 20080742
We studied human cancer cell models in which we detected constitutive activation of ERK. A fraction of active ERK was found to be located in mitochondria in RWPE-2 cells, obtained...