Bingjie Lv
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Explore the profile of Bingjie Lv including associated specialties, affiliations and a list of published articles.
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Articles
30
Citations
321
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Recent Articles
11.
Long Q, Lv B, Jiang S, Lin J
Int J Mol Sci
. 2023 Mar;
24(5).
PMID: 36902000
Cardiovascular disease (CVD) remains the leading cause of mortality globally. Circular RNAs (circRNAs) have attracted extensive attention for their roles in the physiological and pathological processes of various cardiovascular diseases...
12.
Qian T, Qi B, Fei Y, Li J, Luo L, Lv B, et al.
Int Immunopharmacol
. 2023 Jan;
114:109561.
PMID: 36700766
Background: Increased inflammatory exudation caused by endothelium and endothelial junction damage is a typical pathological feature of acute respiratory distress syndrome/acute lung injury (ARDS/ALI). Previous studies have shown that phospholipase...
13.
Yang F, Xia N, Guo S, Zhang J, Liao Y, Tang T, et al.
JACC Basic Transl Sci
. 2022 Nov;
7(9):934-947.
PMID: 36317128
Emerging evidence supports that intestinal microbial metabolite short-chain fatty acids (SCFAs) increase the pool of regulatory T cells (Tregs) in the colonic lamina propria (cLP) and protect against nonintestinal inflammatory...
14.
Li J, Xia N, Li D, Wen S, Qian S, Lu Y, et al.
Adv Sci (Weinh)
. 2022 Mar;
9(9):e2104338.
PMID: 35332699
In addition to maintaining immune tolerance, Foxp3 regulatory T cells (Tregs) perform specialized functions in tissue homeostasis and remodeling. However, whether Tregs in aortic aneurysms have a tissue-specific phenotype and...
15.
Li D, Li J, Liu H, Zhai L, Hu W, Xia N, et al.
FASEB J
. 2022 Feb;
36(3):e22172.
PMID: 35133017
Abdominal aortic aneurysms (AAAs) elicit massive inflammatory leukocyte recruitment to the aorta. CD4 T cells, which include regulatory T cells (Tregs) and conventional T cells (Tconvs), are involved in the...
16.
Lv B, Zhou J, He S, Zheng Y, Yang W, Liu S, et al.
J Vis Exp
. 2022 Feb;
(179).
PMID: 35129168
Acute myocardial infarction is a common cardiovascular disease with high mortality. Myocardial reperfusion injury can counteract the beneficial effects of heart reflow and induce secondary myocardial injury. A simple and...
17.
Jiao J, He S, Wang Y, Lu Y, Gu M, Li D, et al.
Basic Res Cardiol
. 2021 Jul;
116(1):46.
PMID: 34302556
Overactivated inflammatory responses contribute to adverse ventricular remodeling after myocardial infarction (MI). Regulatory B cells (Bregs) are a newly discovered subset of B cells with immunomodulatory roles in many immune...
18.
Zhang M, Zhou T, Liu M, Xia N, Gu M, Tang T, et al.
Biochem Biophys Res Commun
. 2021 Feb;
547:139-147.
PMID: 33610913
Fibrotic scarring is tightly linked to the development of heart failure in patients with post-myocardial infarction (MI). Atypical chemokine receptor 4 (ACKR4) can eliminate chemokines, such as C-C chemokine ligand...
19.
Xia N, Lu Y, Gu M, Li N, Liu M, Jiao J, et al.
Circulation
. 2020 Sep;
142(20):1956-1973.
PMID: 32985264
Background: Regulatory T cells (Tregs), traditionally recognized as potent suppressors of immune response, are increasingly attracting attention because of a second major function: residing in parenchymal tissues and maintaining local...
20.
Interleukin 35 ameliorates myocardial ischemia-reperfusion injury by activating the gp130-STAT3 axis
Zhou X, Xia N, Lv B, Tang T, Nie S, Zhang M, et al.
FASEB J
. 2020 Jan;
34(2):3224-3238.
PMID: 31917470
Myocardial ischemia-reperfusion injury (MIRI) is common clinical complication, which represents significant challenge in the treatment of acute myocardial infarction (AMI) diseases. Interleukin 35 (IL-35) exhibits anti-inflammatory properties via the engagement...