Bilal R Malik
Overview
Explore the profile of Bilal R Malik including associated specialties, affiliations and a list of published articles.
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Articles
8
Citations
326
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0
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Recent Articles
1.
Malik B, Maddison D, Smith G, Peters O
Mol Brain
. 2019 Dec;
12(1):100.
PMID: 31783880
Due to their post-mitotic state, metabolic demands and often large polarised morphology, the function and survival of neurons is dependent on an efficient cellular waste clearance system both for generation...
2.
Higham J, Malik B, Buhl E, Dawson J, Ogier A, Lunnon K, et al.
Front Cell Neurosci
. 2019 Jun;
13:260.
PMID: 31244615
Alzheimer's disease (AD) is the most common form of dementia and is characterized by intracellular neurofibrillary tangles of hyperphosphorylated Tau, including the 0N4R isoform and accumulation of extracellular amyloid beta...
3.
Malik B, Godena V, Whitworth A
Hum Mol Genet
. 2015 Aug;
24(21):6106-17.
PMID: 26251041
Mutations in VPS35 (PARK17) cause autosomal dominant, late onset Parkinson's disease (PD). VPS35 forms a core component of the retromer complex that mediates the retrieval of membrane proteins from endosomes...
4.
Malik B, Hodge J
J Vis Exp
. 2014 Aug;
(90):e50107.
PMID: 25145496
Drosophila have been used in classical conditioning experiments for over 40 years, thus greatly facilitating our understanding of memory, including the elucidation of the molecular mechanisms involved in cognitive diseases....
5.
Malik B, Hodge J
Front Neurosci
. 2014 Jul;
8:178.
PMID: 25009461
Calcium (Ca(2+)) and Calmodulin (CaM)-dependent serine/threonine kinase II (CaMKII) plays a central role in synaptic plasticity and memory due to its ability to phosphorylate itself and regulate its own kinase...
6.
Cavaliere S, Malik B, Hodge J
PLoS One
. 2013 May;
8(4):e62445.
PMID: 23638087
In humans KCNQ2/3 heteromeric channels form an M-current that acts as a brake on neuronal excitability, with mutations causing a form of epilepsy. The M-current has been shown to be...
7.
Malik B, Gillespie J, Hodge J
Front Neural Circuits
. 2013 Apr;
7:52.
PMID: 23543616
Ca(2+)/CaM serine/threonine kinase II (CaMKII) is a central molecule in mechanisms of synaptic plasticity and memory. A vital feature of CaMKII in plasticity is its ability to switch to a...
8.
Emes R, Pocklington A, Anderson C, Bayes A, Collins M, Vickers C, et al.
Nat Neurosci
. 2008 Jun;
11(7):799-806.
PMID: 18536710
Understanding the origins and evolution of synapses may provide insight into species diversity and the organization of the brain. Using comparative proteomics and genomics, we examined the evolution of the...