Bernhard J Mitterauer
Overview
Explore the profile of Bernhard J Mitterauer including associated specialties, affiliations and a list of published articles.
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12
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55
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Recent Articles
1.
Mitterauer B
Cognit Comput
. 2015 Feb;
7:64-73.
PMID: 25685240
A new model of the reticular formation of the brainstem is proposed. It refers to the neuronal and glial cell systems. Thus, it is biomimetically founded. The reticular formation generates...
2.
Mitterauer B
Med Hypotheses
. 2015 Feb;
84(4):315-20.
PMID: 25655220
Based on a logic of balance mechanisms influencing information processing in tripartite synapses are proposed. It is hypothesized that the number of expressed astrocytic receptors determines balanced and imbalanced synaptic...
3.
Mitterauer B
Med Hypotheses
. 2013 Oct;
81(6):1083-7.
PMID: 24169406
The previously proposed hypothesis on the astrocyte mega-domains of autistic savantism is here further elaborated with regard to the faculty of intuition. Two mechanisms may essentially be responsible for the...
4.
Werner G, Mitterauer B
Front Neural Circuits
. 2013 Mar;
7:36.
PMID: 23532509
We examine the interactions and interdependencies between Neuroglia, the Brain-Cell Microenvironment, and the processes commonly subsumed under Neuromodulation. The interactions of the component processes covering a wide spectrum of frequencies...
5.
6.
Mitterauer B
Med Hypotheses
. 2012 Oct;
80(1):17-22.
PMID: 23098371
Individuals with autism who show high abilities are called savants. Whereas in their brains a disconnection in and between neural networks has been identified, savantism is yet poorly understood. Focusing...
7.
Mitterauer B
CNS Neurosci Ther
. 2011 Sep;
17(5):333-44.
PMID: 21951367
Cognitive impairment is a core disorder of the schizophrenia syndrome. Based on glial-neuronal interactions, a pathophysiological model is proposed that could be explanatory for cognitive impairment in schizophrenia. The model...
8.
Mitterauer B, Kofler-Westergren B
Front Psychiatry
. 2011 Jun;
2:15.
PMID: 21647404
A model of glial-neuronal interactions is proposed that could be explanatory for the demyelination identified in brains with schizophrenia. It is based on two hypotheses: (1) that glia-neuron systems are...
9.
Mitterauer B
Med Hypotheses
. 2010 Dec;
76(4):482-5.
PMID: 21159448
The hypothesis is based on glial-neuronal interactions in the cardio-respiratory centre of the brainstem. Recently, it has been experimentally verified that glial cells, especially astrocytes, exert a modulatory function in...
10.
Mitterauer B
CNS Neurosci Ther
. 2010 Jul;
17(5):281-93.
PMID: 20626435
The model of the pathophysiology of bipolar disorder proposed is based on imbalances in tripartite synapses caused by dysregulations of connexin expression in the astrocytic syncytium. If the expression of...