Barbara Ikejiri
Overview
Explore the profile of Barbara Ikejiri including associated specialties, affiliations and a list of published articles.
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Articles
12
Citations
312
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0
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Recent Articles
1.
Gordon I, Lu J, Graves C, Huntoon K, Frerich J, Hanson R, et al.
Mol Cancer Ther
. 2015 May;
14(7):1540-1547.
PMID: 25939762
Protein phosphatase 2A (PP2A) is a tumor suppressor whose function is lost in many cancers. An emerging, though counterintuitive, therapeutic approach is inhibition of PP2A to drive damaged cells through...
2.
Yang C, Wang H, Zhu D, Hong C, Dmitriev P, Zhang C, et al.
Proc Natl Acad Sci U S A
. 2015 Jan;
112(4):1137-42.
PMID: 25583479
Gaucher disease is caused by mutations of the GBA1 gene, which encodes the lysosomal anchored gluococerebrosidase (GCase). GBA1 mutations commonly result in protein misfolding, abnormal chaperone recognition, and premature degradation,...
3.
Yang C, Rahimpour S, Lu J, Pacak K, Ikejiri B, Brady R, et al.
Proc Natl Acad Sci U S A
. 2013 Jan;
110(3):966-71.
PMID: 23277556
Gaucher disease is caused by mutations of the GBA gene that encodes the lysosomal enzyme glucocerebrosidase (GCase). GBA mutations often result in protein misfolding and premature degradation, but usually exert...
4.
Yang C, Iyer R, Yu A, Yong R, Park D, Weil R, et al.
Proc Natl Acad Sci U S A
. 2012 Apr;
109(18):6963-8.
PMID: 22505738
Astrocytes are the most abundant cell of the CNS and demonstrate contact inhibition in which a nonproliferative, nonmotile cellular state is achieved once stable intercellular contacts are formed between mature...
5.
Lu J, Zhuang Z, Song D, Mehta G, Ikejiri B, Mushlin H, et al.
J Neurosurg
. 2009 Dec;
113(2):225-33.
PMID: 20001590
Object: Nuclear receptor corepressor (N-CoR) forms a complex that maintains neural stem cells in an undifferentiated state through transcriptional repression. Recently, it has been shown that N-CoR is overexpressed in...
6.
Li J, Yin C, Okamoto H, Mushlin H, Balgley B, Lee C, et al.
Neuro Oncol
. 2008 Jan;
10(1):45-51.
PMID: 18182627
Dynamic changes in the expression of multiple genes appear to be common features that distinguish transformed cells from their normal counterparts. We compared the proteomic profiles of four glioblastoma multiforme...
7.
Okamoto H, Li J, Vortmeyer A, Jaffe H, Lee Y, Glasker S, et al.
Cancer Res
. 2006 Oct;
66(20):10199-204.
PMID: 17047085
Meningiomas are classified into three groups (benign, atypical, and anaplastic) based on morphologic characteristics. Atypical meningiomas, which are WHO grade 2 tumors, and anaplastic meningiomas, which are WHO grade 3...
8.
Lubensky I, Vortmeyer A, Kim S, Lonser R, Park D, Ikejiri B, et al.
Cell Cycle
. 2006 Feb;
5(4):452-6.
PMID: 16479164
The pathogenesis of de novo glioblastoma multiforme (GBM) is poorly understood and precursor cells are not known. To gain insight into the pathogenesis of GBM we analyzed brains from primates...
9.
Glasker S, Lonser R, Tran M, Ikejiri B, Butman J, Zeng W, et al.
Cancer Res
. 2005 Dec;
65(23):10847-53.
PMID: 16322231
The von Hippel-Lindau (VHL) disease is caused by VHL germ line mutation. Inactivation of the wild-type copy of the VHL gene leads to up-regulation of hypoxic response and tumor formation...
10.
Vogel T, Vortmeyer A, Lubensky I, Lee Y, Furuta M, Ikejiri B, et al.
J Neurosurg
. 2005 Sep;
103(2):284-8.
PMID: 16175858
Object: Von Hippel-Lindau (VHL) disease is characterized by multiple tumors in specific organs. The cell of origin and the reason for the particular organ distribution of the tumors remains unknown....