Azim M Mohamedali
Overview
Explore the profile of Azim M Mohamedali including associated specialties, affiliations and a list of published articles.
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13
Citations
595
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Recent Articles
1.
Kulasekararaj A, Jiang J, Smith A, Mohamedali A, Mian S, Gandhi S, et al.
Blood
. 2014 Aug;
124(17):2698-704.
PMID: 25139356
The distinction between acquired aplastic anemia (AA) and hypocellular myelodysplastic syndrome (hMDS) is often difficult, especially nonsevere AA. We postulated that somatic mutations are present in a subset of AA,...
2.
Kulasekararaj A, Mohamedali A, Mufti G
Br J Haematol
. 2013 Jul;
162(5):587-605.
PMID: 23869491
The advent of novel genomic sequencing technologies has aided the identification of somatically acquired genetic abnormalities up to 80% of myelodysplastic syndrome (MDS) patients. Novel recurrent genetic mutations in pathways...
3.
Mohamedali A, Alkhatabi H, Kulasekararaj A, Shinde S, Mian S, Malik F, et al.
Blood
. 2013 Jun;
122(4):567-70.
PMID: 23760614
Recent studies have shown that more than 80% of bone marrow (BM) samples from patients with myelodysplastic syndrome (MDS) harbor somatic mutations and/or genomic aberrations, which are of diagnostic and...
4.
Gaymes T, Mohamedali A, Patterson M, Matto N, Smith A, Kulasekararaj A, et al.
Haematologica
. 2013 Jan;
98(9):1397-406.
PMID: 23349304
Inactivation of the DNA mismatch repair pathway manifests as microsatellite instability, an accumulation of mutations that drives carcinogenesis. Here, we determined whether microsatellite instability in acute myeloid leukemia and myelodysplastic...
5.
Mian S, Smith A, Kulasekararaj A, Kizilors A, Mohamedali A, Lea N, et al.
Haematologica
. 2013 Jan;
98(7):1058-66.
PMID: 23300180
The recent identification of acquired mutations in key components of the spliceosome machinery strongly implicates abnormalities of mRNA splicing in the pathogenesis of myelodysplastic syndromes. However, questions remain as to...
6.
Kulasekararaj A, Smith A, Mian S, Mohamedali A, Krishnamurthy P, Lea N, et al.
Br J Haematol
. 2013 Jan;
160(5):660-72.
PMID: 23297687
This study aimed to determine the incidence/prognostic impact of TP53 mutation in 318 myelodysplastic syndrome (MDS) patients, and to correlate the changes to cytogenetics, single nucleotide polymorphism array karyotyping and...
7.
Jerez A, Sugimoto Y, Makishima H, Verma A, Jankowska A, Przychodzen B, et al.
Blood
. 2012 May;
119(25):6109-17.
PMID: 22553315
Loss of heterozygosity affecting chromosome 7q is common in acute myeloid leukemia and myelodysplastic syndromes, pointing toward the essential role of this region in disease phenotype and clonal evolution. The...
8.
Westbrook R, Lea N, Mohamedali A, Smith A, Orr D, Roberts L, et al.
Liver Transpl
. 2012 Apr;
18(7):819-27.
PMID: 22467227
Latent myeloproliferative disorders (MPDs) can be identified by Janus kinase 2 (JAK2) mutations in patients with idiopathic Budd-Chiari syndrome (BCS). The incidence and clinical outcomes of JAK2 mutations, novel ten-eleven...
9.
Jerez A, Gondek L, Jankowska A, Makishima H, Przychodzen B, Tiu R, et al.
J Clin Oncol
. 2012 Feb;
30(12):1343-9.
PMID: 22370328
Purpose: Interstitial deletions of chromosome 5q are common in acute myeloid leukemia (AML) and myelodysplastic syndromes (MDS), pointing toward the pathogenic role of this region in disease phenotype and clonal...
10.
Gaken J, Mohamedali A, Jiang J, Malik F, Stangl D, Smith A, et al.
Nucleic Acids Res
. 2012 Feb;
40(10):e75.
PMID: 22323518
MicroRNAs (miRNA) are a class of small RNA molecules that regulate numerous critical cellular processes and bind to partially complementary sequences resulting in down-regulation of their target genes. Due to...