Ariel Contreras-Ferrat
Overview
Explore the profile of Ariel Contreras-Ferrat including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
20
Citations
545
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
11.
Llanos P, Contreras-Ferrat A, Barrientos G, Valencia M, Mears D, Hidalgo C
PLoS One
. 2015 Jun;
10(6):e0129238.
PMID: 26046640
Glucose-stimulated insulin secretion (GSIS) from pancreatic β-cells requires an increase in intracellular free Ca2+ concentration ([Ca2+]). Glucose uptake into β-cells promotes Ca2+ influx and reactive oxygen species (ROS) generation. In...
12.
Pedrozo Z, Criollo A, Battiprolu P, Morales C, Contreras-Ferrat A, Fernandez C, et al.
Circulation
. 2015 Apr;
131(24):2131-42.
PMID: 25888683
Background: L-type calcium channel activity is critical to afterload-induced hypertrophic growth of the heart. However, the mechanisms governing mechanical stress-induced activation of L-type calcium channel activity are obscure. Polycystin-1 (PC-1)...
13.
Llanos P, Contreras-Ferrat A, Georgiev T, Osorio-Fuentealba C, Espinosa A, Hidalgo J, et al.
Am J Physiol Endocrinol Metab
. 2014 Dec;
308(4):E294-305.
PMID: 25491723
Insulin stimulates glucose uptake in adult skeletal muscle by promoting the translocation of GLUT4 glucose transporters to the transverse tubule (T-tubule) membranes, which have particularly high cholesterol levels. We investigated...
14.
Gutierrez T, Parra V, Troncoso R, Pennanen C, Contreras-Ferrat A, Vasquez-Trincado C, et al.
Cell Commun Signal
. 2014 Nov;
12:68.
PMID: 25376904
Background: Cardiac hypertrophy is characterized by alterations in both cardiac bioenergetics and insulin sensitivity. Insulin promotes glucose uptake by cardiomyocytes and its use as a substrate for glycolysis and mitochondrial...
15.
Contreras-Ferrat A, Llanos P, Vasquez C, Espinosa A, Osorio-Fuentealba C, Arias-Calderon M, et al.
J Cell Sci
. 2014 Feb;
127(Pt 9):1911-23.
PMID: 24569874
Insulin signaling includes generation of low levels of H2O2; however, its origin and contribution to insulin-stimulated glucose transport are unknown. We tested the impact of H2O2 on insulin-dependent glucose transport...
16.
Del Campo A, Parra V, Vasquez-Trincado C, Gutierrez T, Morales P, Lopez-Crisosto C, et al.
Am J Physiol Endocrinol Metab
. 2013 Oct;
306(1):E1-E13.
PMID: 24085037
Insulin is a major regulator of glucose metabolism, stimulating its mitochondrial oxidation in skeletal muscle cells. Mitochondria are dynamic organelles that can undergo structural remodeling in order to cope with...
17.
Espinosa A, Campos C, Diaz-Vegas A, Galgani J, Juretic N, Osorio-Fuentealba C, et al.
Int J Mol Sci
. 2013 Aug;
14(8):15740-54.
PMID: 23899788
Insulin resistance is defined as a reduced ability of insulin to stimulate glucose utilization. C57BL/6 mice fed with a high-fat diet (HFD) are a model of insulin resistance. In skeletal...
18.
Wilson C, Contreras-Ferrat A, Venegas N, Osorio-Fuentealba C, Pavez M, Montoya K, et al.
J Cell Physiol
. 2013 Jun;
228(12):2399-407.
PMID: 23757167
Testosterone exerts important effects in the heart. Cardiomyocytes are target cells for androgens, and testosterone induces rapid effects via Ca(2+) release and protein kinase activation and long-term effects via cardiomyocyte...
19.
Jorquera G, Altamirano F, Contreras-Ferrat A, Almarza G, Buvinic S, Jacquemond V, et al.
J Cell Sci
. 2013 Jan;
126(Pt 5):1189-98.
PMID: 23321639
An important pending question in neuromuscular biology is how skeletal muscle cells decipher the stimulation pattern coming from motoneurons to define their phenotype as slow or fast twitch muscle fibers....
20.
Niu W, Bilan P, Ishikura S, Schertzer J, Contreras-Ferrat A, Fu Z, et al.
Am J Physiol Endocrinol Metab
. 2010 Feb;
298(5):E1058-71.
PMID: 20159855
Muscle contraction stimulates glucose uptake acutely to increase energy supply, but suitable cellular models that faithfully reproduce this complex phenomenon are lacking. To this end, we have developed a cellular...