Anita P Merriam
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Explore the profile of Anita P Merriam including associated specialties, affiliations and a list of published articles.
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14
Citations
504
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Recent Articles
1.
Baker P, Kearney J, Gong B, Merriam A, Kuhn D, Porter J, et al.
Neurogenetics
. 2006 Mar;
7(2):81-91.
PMID: 16525850
Dystrophin deficiency leads to the progressive muscle wasting disease Duchenne muscular dystrophy (DMD). Dystrophin-deficient mdx mice are characterized by skeletal muscle weakness and degeneration but they appear outwardly normal in...
2.
Zhou L, Porter J, Cheng G, Gong B, Hatala D, Merriam A, et al.
Neuromuscul Disord
. 2005 Dec;
16(1):32-8.
PMID: 16373085
To address potential regulatory roles of TGF-beta1 in muscle inflammation and fibrosis associated with dystrophin deficiency, we performed quantitative RT-PCR and in situ hybridization to characterize the temporal and spatial...
3.
Porter J, Israel S, Gong B, Merriam A, Feuerman J, Khanna S, et al.
Physiol Genomics
. 2005 Nov;
24(3):264-75.
PMID: 16291736
Skeletal muscles are not created equal. The underutilized concept of muscle allotypes defines distinct muscle groups that differ in their intrinsic capacity to express novel traits when exposed to a...
4.
Sanford J, Edwards J, Mays T, Gong B, Merriam A, Rafael-Fortney J
J Mol Cell Cardiol
. 2005 Feb;
38(2):323-32.
PMID: 15698839
Remodeling of adherens junction, gap junction, and desmosomal proteins at the intercalated discs of cardiomyocytes in heart characterizes several animal models of cardiomyopathy, especially dilated cardiac myopathy (DCM). In this...
5.
Cheng G, Merriam A, Gong B, Leahy P, Khanna S, Porter J
Physiol Genomics
. 2004 May;
18(2):184-95.
PMID: 15138310
Current models in skeletal muscle biology do not fully account for the breadth, causes, and consequences of phenotypic variation among skeletal muscle groups. The muscle allotype concept arose to explain...
6.
Porter J, Merriam A, Leahy P, Gong B, Feuerman J, Cheng G, et al.
Hum Mol Genet
. 2003 Dec;
13(3):257-69.
PMID: 14681298
Mutations in dystrophin are the proximate cause of Duchenne muscular dystrophy (DMD), but pathogenic mechanisms linking the absence of dystrophin from the sarcolemma to myofiber necrosis are not fully known....
7.
Porter J, Merriam A, Gong B, Kasturi S, Zhou X, Hauser K, et al.
J Exp Biol
. 2003 Jul;
206(Pt 17):3101-12.
PMID: 12878677
The M-line and its associated creatine kinase (CK) M-isoform (CK-M) are ubiquitous features of skeletal and cardiac muscle. The M-line maintains myosin myofilaments in register, links the contractile apparatus to...
8.
Porter J, Merriam A, Leahy P, Gong B, Khanna S
Hum Mol Genet
. 2003 Jul;
12(15):1813-21.
PMID: 12874102
Although dystrophin mutations are the proximate cause of Duchenne muscular dystrophy (DMD), interactions among heterogeneous downstream mechanisms may be key phenotypic determinants. Temporal gene expression profiling was used to identify...
9.
Khanna S, Merriam A, Gong B, Leahy P, Porter J
FASEB J
. 2003 Jul;
17(10):1370-2.
PMID: 12832294
Muscle tissue is an elegant model for biologic integration of structure with function and is frequently affected by a variety of inherited diseases. Traditional muscle classes--skeletal, cardiac, and smooth--share basic...
10.
Andrade F, Merriam A, Guo W, Cheng G, McMullen C, Hayess K, et al.
J Appl Physiol (1985)
. 2003 Apr;
95(2):692-9.
PMID: 12716871
The M lines are structural landmarks in striated muscles, necessary for sarcomeric stability and as anchoring sites for the M isoform of creatine kinase (CK-M). These structures, especially prominent in...