Andrew T Knox
Overview
Explore the profile of Andrew T Knox including associated specialties, affiliations and a list of published articles.
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6
Citations
45
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Recent Articles
1.
Knox A, Thompson C, Scott D, Abramova T, Stieve B, Freeman A, et al.
Ann Clin Transl Neurol
. 2025 Jan;
PMID: 39838578
Objective: Interpretation of clinical genetic testing, which identifies a potential genetic etiology in 25% of children with epilepsy, is limited by variants of uncertain significance. Understanding functional consequences of variants...
2.
Jewell T, Carrasco M, Hsu D, Knox A
J Child Neurol
. 2024 Oct;
40(2):116-122.
PMID: 39397495
Neonatal seizures are associated with worsened neurodevelopmental outcomes. Phenobarbital, the only US Food and Drug Administration (FDA)-approved treatment for neonatal seizures, can cause neuronal apoptosis and may worsen neurodevelopmental outcomes....
3.
Knox A, Khakoo Y, Gombolay G
Pediatr Neurol
. 2023 Sep;
148:54-55.
PMID: 37659138
No abstract available.
4.
Swanson L, Hsu D, Ahmed R, Brucker J, Knox A
Epilepsy Behav Rep
. 2021 Apr;
16:100444.
PMID: 33889835
Temporal lobe encephalocele has emerged as a potentially unrecognized cause of drug-resistant temporal lobe epilepsy (TLE) that can be effectively treated with epilepsy surgery. Here we present a case in...
5.
Bradberry M, Courtney N, Dominguez M, Lofquist S, Knox A, Sutton R, et al.
Neuron
. 2020 May;
107(1):52-64.e7.
PMID: 32362337
At neuronal synapses, synaptotagmin-1 (syt1) acts as a Ca sensor that synchronizes neurotransmitter release with Ca influx during action potential firing. Heterozygous missense mutations in syt1 have recently been associated...
6.
Knox A, Glauser T, Tenney J, Lytton W, Holland K
Epilepsia
. 2017 Dec;
59(1):135-145.
PMID: 29265352
Objective: Childhood absence epilepsy (CAE) is a genetic generalized epilepsy syndrome with polygenic inheritance, with genes for γ-aminobutyric acid (GABA) receptors and T-type calcium channels implicated in the disorder. Previous...
7.
Knox A, Powell S, Logan L
J Pediatric Infect Dis Soc
. 2015 Dec;
1(2):157-9.
PMID: 26619169
No abstract available.