Albert Job
Overview
Explore the profile of Albert Job including associated specialties, affiliations and a list of published articles.
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Articles
5
Citations
42
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Recent Articles
1.
Schneider H, Schmitt L, Job A, Lankat-Buttgereit B, Gress T, Buchholz M, et al.
Neoplasia
. 2024 Aug;
57:101038.
PMID: 39128273
The ATR-CHK1 pathway plays a fundamental role in the DNA damage response and is therefore an attractive target in cancer therapy. The antitumorous effect of ATR inhibitors is at least...
2.
Job A, Tatura M, Schafer C, Lutz V, Schneider H, Lankat-Buttgereit B, et al.
Sci Rep
. 2020 Nov;
10(1):18924.
PMID: 33144657
Inhibition of the kinase ATR, a central regulator of the DNA damage response, eliminates subsets of cancer cells in certain tumors. As previously shown, this is at least partly attributable...
3.
Job A, Schmitt L, von Wenserski L, Lankat-Buttgereit B, Gress T, Buchholz M, et al.
Neoplasia
. 2018 Sep;
20(11):1135-1143.
PMID: 30257222
The phosphoinositide 3-kinase-related kinase ATR is a central regulator of the DNA damage response. Its chemical inhibition eliminates subsets of cancer cells in various tumor types. This effect is caused...
4.
Hocke S, Guo Y, Job A, Orth M, Ziesch A, Lauber K, et al.
Oncotarget
. 2016 Jan;
7(6):7080-95.
PMID: 26755646
The phosphoinositide 3-kinase-related kinase ATR represents a central checkpoint regulator and mediator of DNA-repair. Its inhibition selectively eliminates certain subsets of cancer cells in various tumor types, but the underlying...
5.
Brosicke N, Sallouh M, Prior L, Job A, Weberskirch R, Faissner A
Cell Mol Neurobiol
. 2015 Mar;
35(5):741-53.
PMID: 25783630
Glycoproteins of the extracellular matrix (ECM) regulate proliferation, migration, and differentiation in numerous cell lineages. ECM functions are initiated by small peptide sequences embedded in large constituents that are recognized...