Akira Ishisaki
Overview
Explore the profile of Akira Ishisaki including associated specialties, affiliations and a list of published articles.
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80
Citations
766
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Recent Articles
1.
Abe K, Yokota S, Matsumoto S, Ujiie H, Kikuchi E, Satoh K, et al.
J Oral Biosci
. 2025 Jan;
:100609.
PMID: 39755166
Objectives: Temporomandibular joint (TMJ) osteoarthritis (OA) is an inflammatory disease that involves periarthritis of the TMJ and destruction of cartilage tissue in the mandibular condyle. However, the role of proinflammatory...
2.
Yoshida H, Yokota S, Satoh K, Ishisaki A, Chosa N
J Oral Biosci
. 2024 Jan;
66(1):68-75.
PMID: 38266705
Objectives: Cellular differentiation is based on the effects of various growth factors. Transforming growth factor (TGF)-β1 plays a pivotal role in inducing osteogenic differentiation of mesenchymal stem cells (MSCs). In...
3.
Obara N, Kyakumoto S, Yamaguchi S, Yamada H, Ishisaki A, Kamo M
J Oral Biosci
. 2023 Nov;
66(1):151-159.
PMID: 38030062
Objectives: This study aimed to clarify the molecular mechanism underlying the higher invasion and metastasis abilities of LMF4 cells than those of HSC-3 cells by comparing the expression levels of...
4.
Asanuma K, Yokota S, Chosa N, Kamo M, Ibi M, Mayama H, et al.
J Oral Biosci
. 2022 Dec;
65(1):97-103.
PMID: 36584898
Objectives: Temporomandibular joint osteoarthritis (TMJ-OA) is a multifactorial disease caused by inflammation and oxidative stress. It has been hypothesized that mechanical stress-induced injury of TMJ tissues induces the generation of...
5.
Yokota S, Chosa N, Matsumoto S, Satoh K, Ishisaki A
Mol Biol Rep
. 2022 Dec;
50(2):1595-1602.
PMID: 36526849
Background: Temporomandibular joint osteoarthritis (TMJ-OA) causes cartilage degeneration, bone cavitation, and fibrosis of the TMJ. However, the mechanisms underlying the fibroblast-like synoviocyte (FLS)-mediated inflammatory activity in TMJ-OA remain unclear. Methods...
6.
Kozutsumi R, Kuroshima S, Kaneko H, Sasaki M, Ishisaki A, Sawase T
Calcif Tissue Int
. 2021 Aug;
110(1):104-116.
PMID: 34363509
The pathophysiology, histopathology, and immunopathology of bisphosphonate-related osteonecrosis of the jaw (BRONJ) Stage 0 remain unclear. The aim of this study was to investigate the effects of high-dose bisphosphonates on...
7.
Matsumoto S, Yokota S, Chosa N, Kyakumoto S, Kimura H, Kamo M, et al.
Exp Ther Med
. 2020 Aug;
20(3):1967-1974.
PMID: 32782506
Osteoarthritis (OA)-related fibrosis is a possible cause of temporomandibular joint (TMJ) stiffness. However, the molecular mechanisms underlying the fibrogenic activity in fibroblast-like synoviocytes (FLSs) remain to be clarified. The present...
8.
Tamaki S, Kuroshima S, Hayano H, Nakajima K, Kakehashi H, Ishisaki A, et al.
Bone
. 2020 Jul;
141:115560.
PMID: 32730924
Denosumab-related osteonecrosis of the jaw (DRONJ), which mainly occurs in cancer patients receiving anti-receptor activator NF-kappaB ligand (RANKL) antibody, reduces oral health-related quality of life. However, the exact mechanisms of...
9.
Hirano T, Saito D, Yamada H, Ishisaki A, Kamo M
Oncol Lett
. 2020 Jun;
20(1):474-482.
PMID: 32565972
Squamous cell carcinoma (SCC) is the most frequent cancer that develops in the oral cavity. Epithelial-mesenchymal transition (EMT) is known to play an important role in the process of metastasis...
10.
Hayano H, Kuroshima S, Sasaki M, Tamaki S, Inoue M, Ishisaki A, et al.
Bone
. 2020 Mar;
135:115308.
PMID: 32142911
There is limited information about denosumab-related osteonecrosis of the jaw (DRONJ), unlike bisphosphonate-related ONJ (BRONJ). The mode of action is clearly different between denosumab and bisphosphonates. DRONJ occurs mainly following...