A Radhika
Overview
Explore the profile of A Radhika including associated specialties, affiliations and a list of published articles.
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Articles
10
Citations
847
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0
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Recent Articles
1.
Radhika A, Lakshmi J, Ariyanachi K, Sakthivadivel V
Maedica (Bucur)
. 2022 Jun;
17(1):134-142.
PMID: 35733755
Pseudomonas aeruginosa is a leading cause of nosocomial infections and is especially prevalent among patients with burn wounds, cystic fibrosis, acute leukemia, organ transplants and intravenous-drug addiction. Acquired metallo-â-lactamases (MBLs)...
2.
Laheji A, Sakharde S, Chidambaram S, Gondhalekar R, Shankar U, Radhika A
J Contemp Dent Pract
. 2013 Feb;
13(6):925-9.
PMID: 23404029
Adenoameloblastoma or adenomatoid odontogenic tumor (AOT) is an uncommon, benign, epithelial lesion of odontogenic origin. It is a rare benign odontogenic tumor of the jaw affecting mostly young individuals with...
3.
Adusumilli S, Sudhakar P, Mummidi B, Kumar Varma D, Arora S, Radhika A, et al.
J Contemp Dent Pract
. 2013 Feb;
13(6):918-24.
PMID: 23404028
Aim: The present case report describes the importance of understanding of biomechanical and clinical considerations in application of Forsus appliance in correction of class II skeletal malocclusion. Background: Angle's class...
4.
Radhika A, Sudhakaran P
Mol Cell Biochem
. 2012 Oct;
372(1-2):181-90.
PMID: 23054190
Formation of foam cells from macrophages, which are formed by the differentiation of blood-borne monocytes, is a critical early event in atherogenesis. To examine how pre-exposure of monocytes to modified...
5.
Sudhakaran P, Radhika A, Jacob S
Glycoconj J
. 2006 Nov;
24(1):49-55.
PMID: 17115276
Transendothelial migration of monocytes followed by their differentiation into macrophages involves interaction of monocytes with subendothelial matrix. The influence of extracellular matrix on monocyte-macrophage differentiation was studied using an in...
6.
Kandil H, Tanner G, Smalley W, Halter S, Radhika A, DuBois R
Dig Dis Sci
. 2001 May;
46(4):785-9.
PMID: 11330414
Barrett's epithelium is a recognized premalignant condition for esophageal adenocarcinoma. Nonsteroidal antiinflammatory drugs (NSAIDs) decrease the relative risk of colon cancer in humans and the esophageal tumor load in carcinogen-treated...
7.
Shattuck-Brandt R, Varilek G, Radhika A, Yang F, Washington M, DuBois R
Gastroenterology
. 2000 Jan;
118(2):337-45.
PMID: 10648462
Background & Aims: The pathological and molecular changes associated with colitis-associated colorectal cancer and sporadic colorectal cancer are considered to be distinct. Therefore, we have used a mouse model of...
8.
Williams C, Luongo C, Radhika A, Zhang T, Lamps L, Nanney L, et al.
Gastroenterology
. 1996 Oct;
111(4):1134-40.
PMID: 8831610
Background & Aims: Mutations in the APC gene result in an increased propensity to develop intestinal neoplasia; however, a complete understanding of the mechanisms resulting in tumor formation has remained...
9.
DuBois R, Radhika A, Reddy B, Entingh A
Gastroenterology
. 1996 Apr;
110(4):1259-62.
PMID: 8613017
Background & Aims: Multiple studies show that continuous use of nonsteroidal anti-inflammatory drugs (NSAIDs) lowers the risk of colon cancer in humans and carcinogen-treated rodents. One target for NSAIDs is...
10.
Eberhart C, Coffey R, Radhika A, Giardiello F, Ferrenbach S, DuBois R
Gastroenterology
. 1994 Oct;
107(4):1183-8.
PMID: 7926468
Background/aims: Several clinical, epidemiological, and animal studies indicate that nonsteroidal anti-inflammatory drugs (NSAIDs) may alter the incidence of colorectal cancer. A likely target for NSAIDs is cyclooxygenase, a key enzyme...