Reversible Corticospinal Tract Disease Due to Hyperthyroidism

Overview

Journal Arch Neurol

Specialty Neurology

Date 1977 Oct 1

PMID 907539

Citations 12

Authors

C A Garcia

R H Fleming

Affiliations

Soon will be listed here.

Abstract

Corticospinal tract malfunction was seen in a patient with Graves' disease. The absence of intracranial and intraspinal lesions likely to cause the neurologic manifestations and their regression after control of thyroid dysfunction favors a cause-effect relationship. We review and compare similar cases previously reported and discuss the mechanisms.

Citing Articles

The Resting-State Brain Network Functional Connectivity Changes in Patients With Acute Thyrotoxic Myopathy Based on Independent Component Analysis.

Li Y, Ling M, Huang S, Liang X, Qin Y, Luo Z Front Endocrinol (Lausanne). 2022; 13:829411.

PMID: 35399921 PMC: 8986988. DOI: 10.3389/fendo.2022.829411.

Reversible and unilateral corticospinal tract disease secondary to autoimmune free-T3-thyrotoxicosis.

Canepa C, Srinivasan R, Muhith A BMJ Case Rep. 2017; 2017.

PMID: 28784908 PMC: 5747794. DOI: 10.1136/bcr-2017-221322.

Reactivity to Novel Autoantigens in Patients with Coexisting Central Nervous System Demyelinating Disease and Autoimmune Thyroid Disease.

Greer J, Broadley S, Pender M Front Immunol. 2017; 8:514.

PMID: 28533776 PMC: 5420580. DOI: 10.3389/fimmu.2017.00514.

Neurologic complications of nondiabetic endocrine disorders.

Ishii M Continuum (Minneap Minn). 2014; 20:560-79.

PMID: 24893234 PMC: 10564070. DOI: 10.1212/01.CON.0000450966.68828.45.

Acute thyrotoxic bulbar myopathy with encephalopathic behaviour: an uncommon complication of hyperthyroidism.

Boddu N, Badireddi S, Straub K, Schwankhaus J, Jagana R Case Rep Endocrinol. 2013; 2013:369807.

PMID: 23840978 PMC: 3690644. DOI: 10.1155/2013/369807.