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What Happens in Lead Poisoning?

Overview
Specialty General Medicine
Date 1981 Jan 1
PMID 7463387
Citations 2
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References
1.
London G, Nordmann Y, Safar M, METRAL S, Milliez P . Free erythrocyte protoporphyrin level and nerve conduction velocity in end-stage renal disease. Br Med J. 1975; 4(5992):324-5. PMC: 1675160. DOI: 10.1136/bmj.4.5992.324. View

2.
Benson G, George W, Litchfield M, Seaborn D . Biochemical changes during the initial stages of industrial lead exposure. Br J Ind Med. 1976; 33(1):29-35. PMC: 1008099. DOI: 10.1136/oem.33.1.29. View

3.
GOLDBERG A, Paton W, Thompson J . Pharmacology of the porphyrins and porphobilinogen. Br J Pharmacol Chemother. 1954; 9(1):91-4. PMC: 1509378. DOI: 10.1111/j.1476-5381.1954.tb00822.x. View

4.
Jarrett A, Rimington C, Willoughby D . Delta-Aminolaevulic acid and porphyria. Lancet. 1956; 270(6908):125-7. DOI: 10.1016/s0140-6736(56)91091-1. View

5.
PREISKEL D . Chronic lead poisoning: myopathy or neuritis. Ann Phys Med. 1958; 4(8):293-6. DOI: 10.1093/rheumatology/iv.8.293. View