What Happens in Lead Poisoning?
Overview
Overview
Journal
J R Coll Physicians Lond
Specialty
General Medicine
Date
1981 Jan 1
PMID
7463387
Citations
2
Authors
Authors
Affiliations
Affiliations
Soon will be listed here.
Citing Articles
Inhibition of Na+, K+-ATPase activity by delta-aminolevulinic acid.
Russell V, Lamm M, Taljaard J Neurochem Res. 1983; 8(11):1407-15.
PMID: 6140649 DOI: 10.1007/BF00964997.
Cognitive functioning in lead workers.
Stollery B, Banks H, Broadbent D, Lee W Br J Ind Med. 1989; 46(10):698-707.
PMID: 2818958 PMC: 1009851. DOI: 10.1136/oem.46.10.698.
References
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PMC: 1675160.
DOI: 10.1136/bmj.4.5992.324.
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2.
Benson G, George W, Litchfield M, Seaborn D
. Biochemical changes during the initial stages of industrial lead exposure. Br J Ind Med. 1976; 33(1):29-35.
PMC: 1008099.
DOI: 10.1136/oem.33.1.29.
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3.
GOLDBERG A, Paton W, Thompson J
. Pharmacology of the porphyrins and porphobilinogen. Br J Pharmacol Chemother. 1954; 9(1):91-4.
PMC: 1509378.
DOI: 10.1111/j.1476-5381.1954.tb00822.x.
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4.
Jarrett A, Rimington C, Willoughby D
. Delta-Aminolaevulic acid and porphyria. Lancet. 1956; 270(6908):125-7.
DOI: 10.1016/s0140-6736(56)91091-1.
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5.
PREISKEL D
. Chronic lead poisoning: myopathy or neuritis. Ann Phys Med. 1958; 4(8):293-6.
DOI: 10.1093/rheumatology/iv.8.293.
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