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Cerebral Hypoperfusion in the Sudden Infant Death Syndrome? Brainstem Gliosis and Vasculature

Overview
Journal Ann Neurol
Specialty Neurology
Date 1978 Sep 1
PMID 718138
Citations 23
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Abstract

Gliosis is increased in the respiratory control area of the brainstem in victims of sudden infant death syndrome (SIDS), as it is in infants who have died of congenital heart disease. In the latter, the lesions appear to result from hypoxia or ischemia, and studies of the brainstem microvasculature of SIDS victims indicated a close relationship between the gliosis and adjacent vasculature. It is postulated that cerebral hypoperfusion may play a role in SIDS.

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