Low Copper-2 Intake in Switzerland Does Not Result in Lower Incidence of Alzheimer's Disease and Contradicts the Copper-2 Hypothesis
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References
1.
Brewer G
. Avoiding Alzheimer's disease: The important causative role of divalent copper ingestion. Exp Biol Med (Maywood). 2019; 244(2):114-119.
PMC: 6405820.
DOI: 10.1177/1535370219827907.
View
2.
Arens P
. Factors to be considered concerning the corrosion of copper tubes. Eur J Med Res. 1999; 4(6):243-5.
View
3.
Warmlander S, Osterlund N, Wallin C, Wu J, Luo J, Tiiman A
. Metal binding to the amyloid-β peptides in the presence of biomembranes: potential mechanisms of cell toxicity. J Biol Inorg Chem. 2019; 24(8):1189-1196.
DOI: 10.1007/s00775-019-01723-9.
View
4.
Marques-Vidal P, Vollenweider P, Waeber G
. Trends in vitamin, mineral and dietary supplement use in Switzerland. The CoLaus study. Eur J Clin Nutr. 2016; 71(1):122-127.
DOI: 10.1038/ejcn.2016.137.
View
5.
Ceko M, Aitken J, Harris H
. Speciation of copper in a range of food types by X-ray absorption spectroscopy. Food Chem. 2014; 164:50-4.
DOI: 10.1016/j.foodchem.2014.05.018.
View