» Articles » PMID: 31467746

Peritonitis and Subsequent Septic Shock Following Intrauterine Device Removal

Overview
Publisher Wiley
Date 2019 Aug 31
PMID 31467746
Citations 4
Authors
Affiliations
Soon will be listed here.
Abstract

Background: Previous reports have described cases of abscess formation by involving the oral cavity, gastrointestinal tract, and septic thrombophlebitis of the right ovarian vein with subsequent bacteremia and septic shock. Ascending infection from the genital tract to the fallopian tubes resulting in peritonitis from is a rare clinical circumstance where there is minimal information in the literature to guide its diagnosis, management, and expected prognosis.

Case: A 36-year-old G3P0111 developed a tubo-ovarian abscess two weeks after intrauterine device (IUD) removal and then rapidly decompensated with septic shock from peritonitis due to infection. The patient was also newly diagnosed with diabetes and in diabetic ketoacidosis (DKA) on presentation. She received broad-spectrum antibiotic coverage and required two exploratory surgical procedures to obtain source control. Two Interventional Radiology- (IR-) guided drainage procedures were subsequently performed to drain remaining fluid collections. Her recovery involved a prolonged ICU stay. On hospital day seventy-three, after receiving approximately 8 weeks of antibiotics and the above noted procedures the patient was discharged to a subacute rehabilitation facility.

Conclusion: is a highly pathogenic organism once a systemic septic infection has become established that can cause an ascending genital tract infection resulting in tubo-ovarian abscess formation, peritonitis, and septic shock.

Citing Articles

Ruptured Appendiceal Diverticulum Leading to Tubo-Ovarian Abscess in a Non-Sexually Active Woman: A Case Study.

Alenazi H Am J Case Rep. 2024; 25:e945366.

PMID: 39473045 PMC: 11530929. DOI: 10.12659/AJCR.945366.


Necrotizing fasciitis and fatal septic shock associated with .

Rajack F, Medford S, Naab T Autops Case Rep. 2024; 13:e2023467.

PMID: 38213877 PMC: 10782520. DOI: 10.4322/acr.2023.467.


Severe pneumonia with empyema caused by and co-infection: a case report.

Duan Y, Feng W, Shen Y, Li Y, Li N, Chen X J Int Med Res. 2023; 51(11):3000605231210657.

PMID: 37994021 PMC: 10666820. DOI: 10.1177/03000605231210657.


[Pelvic inflammatory disease by Streptococcus constellatus. Clinical experience and a review].

Mora-Palma J, Guillot-Suay V, Sanchez Gila M, Gutierrez-Fernandez J Rev Esp Quimioter. 2020; 33(4):285-288.

PMID: 32515179 PMC: 7374034. DOI: 10.37201/req/020.2020.

References
1.
Herzer C . Toxic shock syndrome: broadening the differential diagnosis. J Am Board Fam Pract. 2001; 14(2):131-6. View

2.
Ness R, Soper D, Holley R, Peipert J, Randall H, Sweet R . Effectiveness of inpatient and outpatient treatment strategies for women with pelvic inflammatory disease: results from the Pelvic Inflammatory Disease Evaluation and Clinical Health (PEACH) Randomized Trial. Am J Obstet Gynecol. 2002; 186(5):929-37. DOI: 10.1067/mob.2002.121625. View

3.
Farley T, Rosenberg M, Rowe P, Chen J, Meirik O . Intrauterine devices and pelvic inflammatory disease: an international perspective. Lancet. 1992; 339(8796):785-8. DOI: 10.1016/0140-6736(92)91904-m. View

4.
Michel P . [Pelvic actinomycosis revealed by pelvic peritonitis]. Ann Chir. 2004; 129(2):96-9. DOI: 10.1016/j.anchir.2003.10.031. View

5.
Ness R, Kip K, Hillier S, Soper D, Stamm C, Sweet R . A cluster analysis of bacterial vaginosis-associated microflora and pelvic inflammatory disease. Am J Epidemiol. 2005; 162(6):585-90. DOI: 10.1093/aje/kwi243. View