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HER2 Signaling Hijacks the Creatine Shuttle to Fuel Breast Cancer Cell Growth

Overview
Journal Cell Metab
Publisher Cell Press
Date 2018 Dec 6
PMID 30517893
Citations 5
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Abstract

Alteration of cell energy metabolism represents a major determinant of cancer progression; however, our understanding of oncogenic mechanisms underlying this rewiring remains elusive. In this issue of Cell Metabolism, Kurmi et al. (2018) show that HER2 signaling promotes ABL-mediated phosphorylation of the mitochondrial creatine kinase (MtCK1), providing ATP to support breast tumor growth.

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