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Increased Mitochondrial Activity in Anthrax-Induced Cell Death

Overview
Journal J Cell Death
Publisher Sage Publications
Specialty Cell Biology
Date 2015 Jul 1
PMID 26124679
Citations 1
Authors
Chi Li
Affiliations
Soon will be listed here.
Abstract

Pathogenesis of anthrax lethal toxin (LT) is attributed to its ability to cause death of infected cells. New work has demonstrated that increase of mitochondrial F1F0 ATPase activity and subsequent depletion of cellular ATP level are critical early events during LT-induced cell death.

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