Owen G, Brenner E
Cell Mol Neurobiol. 2012; 32(6):919-41.
PMID: 22488526
PMC: 11498452.
DOI: 10.1007/s10571-012-9836-0.
Jin I, Kandel E, Hawkins R
Learn Mem. 2011; 18(2):96-102.
PMID: 21245210
PMC: 3032577.
DOI: 10.1101/lm.1949711.
Shobe J, Zhao Y, Stough S, Ye X, Hsuan V, Martin K
Neuron. 2009; 61(1):113-25.
PMID: 19146817
PMC: 4627632.
DOI: 10.1016/j.neuron.2008.10.049.
Hu J, Chen Y, Schacher S
J Neurosci. 2007; 27(33):8927-39.
PMID: 17699674
PMC: 6672177.
DOI: 10.1523/JNEUROSCI.2322-07.2007.
Zhao Y, Leal K, Abi-Farah C, Martin K, Sossin W, Klein M
J Neurosci. 2006; 26(34):8847-56.
PMID: 16928874
PMC: 6674363.
DOI: 10.1523/JNEUROSCI.1919-06.2006.
Two signaling pathways regulate the expression and secretion of a neuropeptide required for long-term facilitation in Aplysia.
Hu J, Wu F, Schacher S
J Neurosci. 2006; 26(3):1026-35.
PMID: 16421322
PMC: 6675361.
DOI: 10.1523/JNEUROSCI.4258-05.2006.
Synaptic facilitation and behavioral dishabituation in Aplysia: dependence on release of Ca2+ from postsynaptic intracellular stores, postsynaptic exocytosis, and modulation of postsynaptic AMPA receptor efficacy.
Li Q, Roberts A, Glanzman D
J Neurosci. 2005; 25(23):5623-37.
PMID: 15944390
PMC: 6724986.
DOI: 10.1523/JNEUROSCI.5305-04.2005.
Coregulation of glutamate uptake and long-term sensitization in Aplysia.
Khabour O, Levenson J, Lyons L, Kategaya L, Chin J, Byrne J
J Neurosci. 2004; 24(40):8829-37.
PMID: 15470149
PMC: 6729961.
DOI: 10.1523/JNEUROSCI.2167-04.2004.
Intermediate-term memory for site-specific sensitization in aplysia is maintained by persistent activation of protein kinase C.
Sutton M, Bagnall M, Sharma S, Shobe J, Carew T
J Neurosci. 2004; 24(14):3600-9.
PMID: 15071108
PMC: 6729755.
DOI: 10.1523/JNEUROSCI.1134-03.2004.
Multiple serotonergic mechanisms contributing to sensitization in aplysia: evidence of diverse serotonin receptor subtypes.
Barbas D, DesGroseillers L, Castellucci V, Carew T, Marinesco S
Learn Mem. 2003; 10(5):373-86.
PMID: 14557610
PMC: 218003.
DOI: 10.1101/lm.66103.
Ca2+-independent protein kinase C Apl II mediates the serotonin-induced facilitation at depressed aplysia sensorimotor synapses.
Manseau F, Fan X, Hueftlein T, Sossin W, Castellucci V
J Neurosci. 2001; 21(4):1247-56.
PMID: 11160395
PMC: 6762221.
Inflammation causes a long-term hyperexcitability in the nociceptive sensory neurons of Aplysia.
Farr M, Mathews J, Zhu D, Ambron R
Learn Mem. 1999; 6(3):331-40.
PMID: 10492014
PMC: 311296.
Involvement of protein kinase C in 5-HT-stimulated ciliary activity in Helisoma trivolvis embryos.
Christopher K, Young K, Chang J, Goldberg J
J Physiol. 1999; 515 ( Pt 2):511-22.
PMID: 10050017
PMC: 2269170.
DOI: 10.1111/j.1469-7793.1999.511ac.x.
Binding of serotonin to receptors at multiple sites is required for structural plasticity accompanying long-term facilitation of Aplysia sensorimotor synapses.
Sun Z, Schacher S
J Neurosci. 1998; 18(11):3991-4000.
PMID: 9592080
PMC: 6792818.
Voltage gated calcium channels in molluscs: classification, Ca2+ dependent inactivation, modulation and functional roles.
Kits K, Mansvelder H
Invert Neurosci. 1996; 2(1):9-34.
PMID: 9372153
DOI: 10.1007/BF02336657.
Modulation of ion currents and regulation of transmitter release in short-term synaptic plasticity: the rise and fall of the action potential.
Klein M
Invert Neurosci. 1995; 1(1):15-24.
PMID: 9372129
DOI: 10.1007/BF02331828.
Differential distribution of functional receptors for neuromodulators evoking short-term heterosynaptic plasticity in Aplysia sensory neurons.
Sun Z, Kauderer B, Schacher S
J Neurosci. 1996; 16(23):7540-9.
PMID: 8922410
PMC: 6579104.
Mechanisms of antagonistic action of internal Ca2+ on serotonin-induced potentiation of Ca2+ currents in Helix neurones.
Kostyuk P, Lukyanetz E
Pflugers Arch. 1993; 424(1):73-83.
PMID: 7688896
DOI: 10.1007/BF00375104.
Selective translocation of protein kinase C-delta in PC12 cells during nerve growth factor-induced neuritogenesis.
ODriscoll K, Teng K, Fabbro D, Greene L, Weinstein I
Mol Biol Cell. 1995; 6(4):449-58.
PMID: 7626808
PMC: 301203.
DOI: 10.1091/mbc.6.4.449.
cAMP modulates multiple K+ currents, increasing spike duration and excitability in Aplysia sensory neurons.
Goldsmith B, Abrams T
Proc Natl Acad Sci U S A. 1992; 89(23):11481-5.
PMID: 1333612
PMC: 50575.
DOI: 10.1073/pnas.89.23.11481.