Dominic K, Schmidt A, Granzier H, Campbell K, Stelzer J
Front Physiol. 2024; 15:1512550.
PMID: 39726859
PMC: 11669688.
DOI: 10.3389/fphys.2024.1512550.
Zimmermann H, Macintosh B, Dal Pupo J
Sports Med. 2024; 55(1):37-47.
PMID: 39543073
DOI: 10.1007/s40279-024-02140-y.
Lewalle A, Milburn G, Campbell K, Niederer S
Biophys J. 2024; 123(18):2996-3009.
PMID: 38807364
PMC: 11428202.
DOI: 10.1016/j.bpj.2024.05.025.
Da Silva Costa A, Ghouri I, Johnston A, McGlynn K, McNair A, Bowman P
Cell Biochem Funct. 2023; 41(8):1147-1161.
PMID: 37665041
PMC: 10947300.
DOI: 10.1002/cbf.3847.
Zechini L, Camilleri-Brennan J, Walsh J, Beaven R, Moran O, Hartley P
Front Physiol. 2022; 13:1003999.
PMID: 36187790
PMC: 9515499.
DOI: 10.3389/fphys.2022.1003999.
High hydrostatic pressure induces slow contraction in mouse cardiomyocytes.
Yamaguchi Y, Nishiyama M, Kai H, Kaneko T, Kaihara K, Iribe G
Biophys J. 2022; 121(17):3286-3294.
PMID: 35841143
PMC: 9463647.
DOI: 10.1016/j.bpj.2022.07.016.
Small Angle X-ray Diffraction as a Tool for Structural Characterization of Muscle Disease.
Ma W, Irving T
Int J Mol Sci. 2022; 23(6).
PMID: 35328477
PMC: 8949570.
DOI: 10.3390/ijms23063052.
GSK-3β Localizes to the Cardiac Z-Disc to Maintain Length Dependent Activation.
Stachowski-Doll M, Papadaki M, Martin T, Ma W, Gong H, Shao S
Circ Res. 2022; 130(6):871-886.
PMID: 35168370
PMC: 8930626.
DOI: 10.1161/CIRCRESAHA.121.319491.
Frank-Starling mechanism, fluid responsiveness, and length-dependent activation: Unravelling the multiscale behaviors with an in silico analysis.
Kosta S, Dauby P
PLoS Comput Biol. 2021; 17(10):e1009469.
PMID: 34634040
PMC: 8504729.
DOI: 10.1371/journal.pcbi.1009469.
Physical Activity Engagement Worsens Health Outcomes and Limits Exercise Capacity in Growth-restricted Mice.
Ferguson D, Leszczynski E, McPeek A, Pendergrast L, Visker J, Triplett A
Med Sci Sports Exerc. 2021; 53(8):1561-1571.
PMID: 34261989
PMC: 10797723.
DOI: 10.1249/MSS.0000000000002620.
Neuregulins: protective and reparative growth factors in multiple forms of cardiovascular disease.
Geissler A, Ryzhov S, Sawyer D
Clin Sci (Lond). 2020; 134(19):2623-2643.
PMID: 33063822
PMC: 7557502.
DOI: 10.1042/CS20200230.
Semi-mechanistic modelling platform to assess cardiac contractility and haemodynamics in preclinical cardiovascular safety profiling of new molecular entities.
Venkatasubramanian R, Collins T, Lesko L, Mettetal J, Trame M
Br J Pharmacol. 2020; 177(15):3568-3590.
PMID: 32335903
PMC: 7348097.
DOI: 10.1111/bph.15079.
Model order reduction for left ventricular mechanics via congruency training.
Di Achille P, Parikh J, Khamzin S, Solovyova O, Kozloski J, Gurev V
PLoS One. 2020; 15(1):e0219876.
PMID: 31905197
PMC: 6944464.
DOI: 10.1371/journal.pone.0219876.
In vivo elongation of thin filaments results in heart failure.
Mi-Mi L, Farman G, Mayfield R, Strom J, Chu M, Pappas C
PLoS One. 2020; 15(1):e0226138.
PMID: 31899774
PMC: 6941805.
DOI: 10.1371/journal.pone.0226138.
Cardiomyopathy mutation (F88L) in troponin T abolishes length dependency of myofilament Ca sensitivity.
Reda S, Chandra M
J Gen Physiol. 2018; 150(6):809-819.
PMID: 29776992
PMC: 5987878.
DOI: 10.1085/jgp.201711974.
Naturally Engineered Maturation of Cardiomyocytes.
Scuderi G, Butcher J
Front Cell Dev Biol. 2017; 5:50.
PMID: 28529939
PMC: 5418234.
DOI: 10.3389/fcell.2017.00050.
Insights and Challenges of Multi-Scale Modeling of Sarcomere Mechanics in cTn and Tm DCM Mutants-Genotype to Cellular Phenotype.
Dewan S, McCabe K, Regnier M, McCulloch A
Front Physiol. 2017; 8:151.
PMID: 28352236
PMC: 5348544.
DOI: 10.3389/fphys.2017.00151.
Myosin MgADP Release Rate Decreases as Sarcomere Length Increases in Skinned Rat Soleus Muscle Fibers.
Fenwick A, Leighton S, Tanner B
Biophys J. 2016; 111(9):2011-2023.
PMID: 27806282
PMC: 5103013.
DOI: 10.1016/j.bpj.2016.09.024.
Thin filament length in the cardiac sarcomere varies with sarcomere length but is independent of titin and nebulin.
Kolb J, Li F, Methawasin M, Adler M, Escobar Y, Nedrud J
J Mol Cell Cardiol. 2016; 97:286-94.
PMID: 27139341
PMC: 5584371.
DOI: 10.1016/j.yjmcc.2016.04.013.
Nanotopography-Induced Structural Anisotropy and Sarcomere Development in Human Cardiomyocytes Derived from Induced Pluripotent Stem Cells.
Carson D, Hnilova M, Yang X, Nemeth C, Tsui J, Smith A
ACS Appl Mater Interfaces. 2016; 8(34):21923-32.
PMID: 26866596
PMC: 5681855.
DOI: 10.1021/acsami.5b11671.